CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia

T-cell prolymphocytic leukemia (T-PLL) is a poor prognostic disease with very limited options of efficient therapies. Most patients are refractory to chemotherapies and despite high response rates after alemtuzumab, virtually all patients relapse. Therefore, there is an unmet medical need for novel...

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Autores: Cuesta-Mateos, Carlos, Fuentes, Patricia, Schrader, Alexandra, Juárez-Sánchez, Raquel, Loscertales, Javier, Mateu-Alberoa, Tamara, Vega-Piris, Lorena, Espartero-Santos, Marina, Marcos-Jiménez, Ana, Sánchez-López, Blanca A., Pérez-García, Yaiza, Jungherz, Dennis, Oberbeckmann, Sebastian, Wahnshaffe, Linus, Andersson, Emma I., Mustjoki, Satu, Faber, Edgar, Urzainqui, Ana, Fresno, Manuel, Stamatakis, Konstantinos, Alfranca, Arántzazu, Terrón, Fernando, Herling, Marco, Toribio, María Luisa, Muñoz-Calleja, Cecilia
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2020
País:España
Institución:Consejo Superior de Investigaciones Científicas (CSIC)
Repositorio:DIGITAL.CSIC. Repositorio Institucional del CSIC
OAI Identifier:oai:digital.csic.es:10261/235552
Acceso en línea:http://hdl.handle.net/10261/235552
Access Level:acceso abierto
Palabra clave:CCR7
T-PLL
mAb
T-cell lymphomas
Immunotherapy
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spelling CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemiaCuesta-Mateos, CarlosFuentes, PatriciaSchrader, AlexandraJuárez-Sánchez, RaquelLoscertales, JavierMateu-Alberoa, TamaraVega-Piris, LorenaEspartero-Santos, MarinaMarcos-Jiménez, AnaSánchez-López, Blanca A.Pérez-García, YaizaJungherz, DennisOberbeckmann, SebastianWahnshaffe, LinusAndersson, Emma I.Mustjoki, SatuFaber, EdgarUrzainqui, AnaFresno, ManuelStamatakis, KonstantinosAlfranca, ArántzazuTerrón, FernandoHerling, MarcoToribio, María LuisaMuñoz-Calleja, CeciliaCCR7T-PLLmAbT-cell lymphomasImmunotherapyT-cell prolymphocytic leukemia (T-PLL) is a poor prognostic disease with very limited options of efficient therapies. Most patients are refractory to chemotherapies and despite high response rates after alemtuzumab, virtually all patients relapse. Therefore, there is an unmet medical need for novel therapies in T-PLL. As the chemokine receptor CCR7 is a molecule expressed in a wide range of malignancies and relevant in many tumor processes, the present study addressed the biologic role of this receptor in T-PLL. Furthermore, we elucidated the mechanisms of action mediated by an anti-CCR7 monoclonal antibody (mAb) and evaluated whether its anti-tumor activity would warrant development towards clinical applications in T-PLL. Our results demonstrate that CCR7 is a prognostic biomarker for overall survival in T-PLL patients and a functional receptor involved in the migration, invasion, and survival of leukemic cells. Targeting CCR7 with a mAb inhibited ligand-mediated signaling pathways and induced tumor cell killing in primary samples. In addition, directing antibodies against CCR7 was highly effective in T-cell leukemia xenograft models. Together, these findings make CCR7 an attractive molecule for novel mAb-based therapeutic applications in T-PLL, a disease where recent drug screen efforts and studies addressing new compounds have focused on chemotherapy or small molecules.(Ministerio de Economía y Competitividad, Spain). MH was funded by the German Research Foundation (DFG; HE3553/4–2) as part of the collaborative research group on mature T-cell lymphomas, “CONTROL-T” (FOR1961). The José Carreras Leukemia Foundation (03F/2016), the Köln Fortune Program, and the Fritz Thyssen foundation (10.15.2.034MN) supported MH and AS. The Köln Fortune Program also supported LW. SM was supported by the Finnish Cancer Organizations, the Finnish Cancer Institute, Academy of Finland and the Sigrid Juselius FoundationBioMed CentralConsejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]2021202120202021info:eu-repo/semantics/articlehttp://purl.org/coar/resource_type/c_6501Publisher's versioninfo:eu-repo/semantics/publishedVersionhttp://hdl.handle.net/10261/235552reponame:DIGITAL.CSIC. Repositorio Institucional del CSICinstname:Consejo Superior de Investigaciones Científicas (CSIC)Ingléshttp://dx.doi.org/10.1186/s40364-020-00234-zSíinfo:eu-repo/semantics/openAccessoai:digital.csic.es:10261/2355522026-05-22T06:33:51Z
dc.title.none.fl_str_mv CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia
title CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia
spellingShingle CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia
Cuesta-Mateos, Carlos
CCR7
T-PLL
mAb
T-cell lymphomas
Immunotherapy
title_short CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia
title_full CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia
title_fullStr CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia
title_full_unstemmed CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia
title_sort CCR7 as a novel therapeutic target in t-cell PROLYMPHOCYTIC leukemia
dc.creator.none.fl_str_mv Cuesta-Mateos, Carlos
Fuentes, Patricia
Schrader, Alexandra
Juárez-Sánchez, Raquel
Loscertales, Javier
Mateu-Alberoa, Tamara
Vega-Piris, Lorena
Espartero-Santos, Marina
Marcos-Jiménez, Ana
Sánchez-López, Blanca A.
Pérez-García, Yaiza
Jungherz, Dennis
Oberbeckmann, Sebastian
Wahnshaffe, Linus
Andersson, Emma I.
Mustjoki, Satu
Faber, Edgar
Urzainqui, Ana
Fresno, Manuel
Stamatakis, Konstantinos
Alfranca, Arántzazu
Terrón, Fernando
Herling, Marco
Toribio, María Luisa
Muñoz-Calleja, Cecilia
author Cuesta-Mateos, Carlos
author_facet Cuesta-Mateos, Carlos
Fuentes, Patricia
Schrader, Alexandra
Juárez-Sánchez, Raquel
Loscertales, Javier
Mateu-Alberoa, Tamara
Vega-Piris, Lorena
Espartero-Santos, Marina
Marcos-Jiménez, Ana
Sánchez-López, Blanca A.
Pérez-García, Yaiza
Jungherz, Dennis
Oberbeckmann, Sebastian
Wahnshaffe, Linus
Andersson, Emma I.
Mustjoki, Satu
Faber, Edgar
Urzainqui, Ana
Fresno, Manuel
Stamatakis, Konstantinos
Alfranca, Arántzazu
Terrón, Fernando
Herling, Marco
Toribio, María Luisa
Muñoz-Calleja, Cecilia
author_role author
author2 Fuentes, Patricia
Schrader, Alexandra
Juárez-Sánchez, Raquel
Loscertales, Javier
Mateu-Alberoa, Tamara
Vega-Piris, Lorena
Espartero-Santos, Marina
Marcos-Jiménez, Ana
Sánchez-López, Blanca A.
Pérez-García, Yaiza
Jungherz, Dennis
Oberbeckmann, Sebastian
Wahnshaffe, Linus
Andersson, Emma I.
Mustjoki, Satu
Faber, Edgar
Urzainqui, Ana
Fresno, Manuel
Stamatakis, Konstantinos
Alfranca, Arántzazu
Terrón, Fernando
Herling, Marco
Toribio, María Luisa
Muñoz-Calleja, Cecilia
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Consejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]
dc.subject.none.fl_str_mv CCR7
T-PLL
mAb
T-cell lymphomas
Immunotherapy
topic CCR7
T-PLL
mAb
T-cell lymphomas
Immunotherapy
description T-cell prolymphocytic leukemia (T-PLL) is a poor prognostic disease with very limited options of efficient therapies. Most patients are refractory to chemotherapies and despite high response rates after alemtuzumab, virtually all patients relapse. Therefore, there is an unmet medical need for novel therapies in T-PLL. As the chemokine receptor CCR7 is a molecule expressed in a wide range of malignancies and relevant in many tumor processes, the present study addressed the biologic role of this receptor in T-PLL. Furthermore, we elucidated the mechanisms of action mediated by an anti-CCR7 monoclonal antibody (mAb) and evaluated whether its anti-tumor activity would warrant development towards clinical applications in T-PLL. Our results demonstrate that CCR7 is a prognostic biomarker for overall survival in T-PLL patients and a functional receptor involved in the migration, invasion, and survival of leukemic cells. Targeting CCR7 with a mAb inhibited ligand-mediated signaling pathways and induced tumor cell killing in primary samples. In addition, directing antibodies against CCR7 was highly effective in T-cell leukemia xenograft models. Together, these findings make CCR7 an attractive molecule for novel mAb-based therapeutic applications in T-PLL, a disease where recent drug screen efforts and studies addressing new compounds have focused on chemotherapy or small molecules.
publishDate 2020
dc.date.none.fl_str_mv 2020
2021
2021
2021
dc.type.none.fl_str_mv info:eu-repo/semantics/article
http://purl.org/coar/resource_type/c_6501
Publisher's version
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/10261/235552
url http://hdl.handle.net/10261/235552
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv http://dx.doi.org/10.1186/s40364-020-00234-z

dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.publisher.none.fl_str_mv BioMed Central
publisher.none.fl_str_mv BioMed Central
dc.source.none.fl_str_mv reponame:DIGITAL.CSIC. Repositorio Institucional del CSIC
instname:Consejo Superior de Investigaciones Científicas (CSIC)
instname_str Consejo Superior de Investigaciones Científicas (CSIC)
reponame_str DIGITAL.CSIC. Repositorio Institucional del CSIC
collection DIGITAL.CSIC. Repositorio Institucional del CSIC
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