Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics

Abstract Acute myeloid leukemia patients with normal cytogenetics (CN-AML) account for almost half of AML cases. We aimed to study the frequency and relationship of a wide range of genes previously reported as mutated in AML (ASXL1, NPM1, FLT3, TET2, IDH1/2, RUNX1, DNMT3A, NRAS, JAK2, WT1, CBL, SF3B...

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Authors: Boultwood, J. (Jacqueline)|||/items/4e3a6660-3a20-426e-aba2-94bdbb24b6e8, Wainscoat, J.S. (James S.)|||/items/2c893d2c-7d50-4f21-9b29-728dcc2fa011, Calasanz-Abinzano, M.J. (Maria Jose)|||/items/a1f10f5c-06ce-47eb-bfd8-91fb972d8086, Prosper-Cardoso, F. (Felipe)|||/items/3d1b0b82-06c3-4e63-8280-e903dc4dc0c1, Aguirre-Ena, X. (Xabier)|||/items/2a000d9c-cb5c-4734-a32c-4fef79998c86, Odero, M.D. (Maria Dolores)|||/items/6679bbec-f7dd-480c-a44c-44d7c8493251, McDonald, E.J. (Emma Jane)|||/items/ec0504d2-270e-400a-b9e1-29ca0786f4ad, Killick, S. (Sally)|||/items/c07eb5b9-2a68-430e-bb08-d9d303598e92, Perez, C. (Cristina)|||/items/290e09cd-b084-4cdd-b63e-84762b2d923f, Kondo, T. (Toshinori)|||/items/c7a125af-b58d-42f7-8914-38a5f3062e84, Larrayoz-Ilundáin, M.J. (María José)|||/items/7e29cbb9-798d-4830-bfcf-729a99e05d02, Davies, C. (Carwyn)|||/items/c1de2fdf-b499-47ae-a9e1-a21ffd0bb595, Pellagatti, A. (Andrea)|||/items/a8dd726b-6248-4850-9885-ea609811ceb9, Yip, B.H. (Bon Ham)|||/items/33607d98-632a-4d9b-8863-a9380a676ebb, Fernandez-Mercado, M. (Marta)|||/items/9919a3c8-2c8c-4d98-9b5b-57864191c2bc
Format: article
Publication Date:2012
Country:España
Institution:Universidad de Navarra
Repository:Dadun. Depósito Académico Digital de la Universidad de Navarra
Language:English
OAI Identifier:oai:dadun.unav.edu:10171/41454
Online Access:https://hdl.handle.net/10171/41454
Access Level:Open access
Keyword:Mutation
Acute Myeloid Leukemia
Normal Cytogenetics
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spelling Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogeneticsBoultwood, J. (Jacqueline)|||/items/4e3a6660-3a20-426e-aba2-94bdbb24b6e8Wainscoat, J.S. (James S.)|||/items/2c893d2c-7d50-4f21-9b29-728dcc2fa011Calasanz-Abinzano, M.J. (Maria Jose)|||/items/a1f10f5c-06ce-47eb-bfd8-91fb972d8086Prosper-Cardoso, F. (Felipe)|||/items/3d1b0b82-06c3-4e63-8280-e903dc4dc0c1Aguirre-Ena, X. (Xabier)|||/items/2a000d9c-cb5c-4734-a32c-4fef79998c86Odero, M.D. (Maria Dolores)|||/items/6679bbec-f7dd-480c-a44c-44d7c8493251McDonald, E.J. (Emma Jane)|||/items/ec0504d2-270e-400a-b9e1-29ca0786f4adKillick, S. (Sally)|||/items/c07eb5b9-2a68-430e-bb08-d9d303598e92Perez, C. (Cristina)|||/items/290e09cd-b084-4cdd-b63e-84762b2d923fKondo, T. (Toshinori)|||/items/c7a125af-b58d-42f7-8914-38a5f3062e84Larrayoz-Ilundáin, M.J. (María José)|||/items/7e29cbb9-798d-4830-bfcf-729a99e05d02Davies, C. (Carwyn)|||/items/c1de2fdf-b499-47ae-a9e1-a21ffd0bb595Pellagatti, A. (Andrea)|||/items/a8dd726b-6248-4850-9885-ea609811ceb9Yip, B.H. (Bon Ham)|||/items/33607d98-632a-4d9b-8863-a9380a676ebbFernandez-Mercado, M. (Marta)|||/items/9919a3c8-2c8c-4d98-9b5b-57864191c2bcMutationAcute Myeloid LeukemiaNormal CytogeneticsAbstract Acute myeloid leukemia patients with normal cytogenetics (CN-AML) account for almost half of AML cases. We aimed to study the frequency and relationship of a wide range of genes previously reported as mutated in AML (ASXL1, NPM1, FLT3, TET2, IDH1/2, RUNX1, DNMT3A, NRAS, JAK2, WT1, CBL, SF3B1, TP53, KRAS and MPL) in a series of 84 CN-AML cases. The most frequently mutated genes in primary cases were NPM1 (60.8%) and FLT3 (50.0%), and in secondary cases ASXL1 (48.5%) and TET2 (30.3%). We showed that 85% of CN-AML patients have mutations in at least one of ASXL1, NPM1, FLT3, TET2, IDH1/2 and/or RUNX1. Serial samples from 19 MDS/CMML cases that progressed to AML were analyzed for ASXL1/TET2/IDH1/2 mutations; seventeen cases presented mutations of at least one of these genes. However, there was no consistent pattern in mutation acquisition during disease progression. This report concerns the analysis of the largest number of gene mutations in CN-AML studied to date, and provides insight into the mutational profile of CN-AMLDadun. Depósito Académico Digital Universidad de Navarra20162016-08-1220122012-01-0120122012-01-01journal articlehttp://purl.org/coar/resource_type/c_6501info:eu-repo/semantics/articleapplication/pdfhttps://hdl.handle.net/10171/41454reponame:Dadun. Depósito Académico Digital de la Universidad de Navarrainstname:Universidad de NavarraInglésengopen accesshttp://purl.org/coar/access_right/c_abf2info:eu-repo/semantics/openAccessoai:dadun.unav.edu:10171/414542026-06-21T12:47:57Z
dc.title.none.fl_str_mv Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics
title Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics
spellingShingle Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics
Boultwood, J. (Jacqueline)|||/items/4e3a6660-3a20-426e-aba2-94bdbb24b6e8
Mutation
Acute Myeloid Leukemia
Normal Cytogenetics
title_short Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics
title_full Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics
title_fullStr Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics
title_full_unstemmed Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics
title_sort Mutation patterns of 16 genes in primary and secondary acute myeloid leukemia (AML) with normal cytogenetics
dc.creator.none.fl_str_mv Boultwood, J. (Jacqueline)|||/items/4e3a6660-3a20-426e-aba2-94bdbb24b6e8
Wainscoat, J.S. (James S.)|||/items/2c893d2c-7d50-4f21-9b29-728dcc2fa011
Calasanz-Abinzano, M.J. (Maria Jose)|||/items/a1f10f5c-06ce-47eb-bfd8-91fb972d8086
Prosper-Cardoso, F. (Felipe)|||/items/3d1b0b82-06c3-4e63-8280-e903dc4dc0c1
Aguirre-Ena, X. (Xabier)|||/items/2a000d9c-cb5c-4734-a32c-4fef79998c86
Odero, M.D. (Maria Dolores)|||/items/6679bbec-f7dd-480c-a44c-44d7c8493251
McDonald, E.J. (Emma Jane)|||/items/ec0504d2-270e-400a-b9e1-29ca0786f4ad
Killick, S. (Sally)|||/items/c07eb5b9-2a68-430e-bb08-d9d303598e92
Perez, C. (Cristina)|||/items/290e09cd-b084-4cdd-b63e-84762b2d923f
Kondo, T. (Toshinori)|||/items/c7a125af-b58d-42f7-8914-38a5f3062e84
Larrayoz-Ilundáin, M.J. (María José)|||/items/7e29cbb9-798d-4830-bfcf-729a99e05d02
Davies, C. (Carwyn)|||/items/c1de2fdf-b499-47ae-a9e1-a21ffd0bb595
Pellagatti, A. (Andrea)|||/items/a8dd726b-6248-4850-9885-ea609811ceb9
Yip, B.H. (Bon Ham)|||/items/33607d98-632a-4d9b-8863-a9380a676ebb
Fernandez-Mercado, M. (Marta)|||/items/9919a3c8-2c8c-4d98-9b5b-57864191c2bc
author Boultwood, J. (Jacqueline)|||/items/4e3a6660-3a20-426e-aba2-94bdbb24b6e8
author_facet Boultwood, J. (Jacqueline)|||/items/4e3a6660-3a20-426e-aba2-94bdbb24b6e8
Wainscoat, J.S. (James S.)|||/items/2c893d2c-7d50-4f21-9b29-728dcc2fa011
Calasanz-Abinzano, M.J. (Maria Jose)|||/items/a1f10f5c-06ce-47eb-bfd8-91fb972d8086
Prosper-Cardoso, F. (Felipe)|||/items/3d1b0b82-06c3-4e63-8280-e903dc4dc0c1
Aguirre-Ena, X. (Xabier)|||/items/2a000d9c-cb5c-4734-a32c-4fef79998c86
Odero, M.D. (Maria Dolores)|||/items/6679bbec-f7dd-480c-a44c-44d7c8493251
McDonald, E.J. (Emma Jane)|||/items/ec0504d2-270e-400a-b9e1-29ca0786f4ad
Killick, S. (Sally)|||/items/c07eb5b9-2a68-430e-bb08-d9d303598e92
Perez, C. (Cristina)|||/items/290e09cd-b084-4cdd-b63e-84762b2d923f
Kondo, T. (Toshinori)|||/items/c7a125af-b58d-42f7-8914-38a5f3062e84
Larrayoz-Ilundáin, M.J. (María José)|||/items/7e29cbb9-798d-4830-bfcf-729a99e05d02
Davies, C. (Carwyn)|||/items/c1de2fdf-b499-47ae-a9e1-a21ffd0bb595
Pellagatti, A. (Andrea)|||/items/a8dd726b-6248-4850-9885-ea609811ceb9
Yip, B.H. (Bon Ham)|||/items/33607d98-632a-4d9b-8863-a9380a676ebb
Fernandez-Mercado, M. (Marta)|||/items/9919a3c8-2c8c-4d98-9b5b-57864191c2bc
author_role author
author2 Wainscoat, J.S. (James S.)|||/items/2c893d2c-7d50-4f21-9b29-728dcc2fa011
Calasanz-Abinzano, M.J. (Maria Jose)|||/items/a1f10f5c-06ce-47eb-bfd8-91fb972d8086
Prosper-Cardoso, F. (Felipe)|||/items/3d1b0b82-06c3-4e63-8280-e903dc4dc0c1
Aguirre-Ena, X. (Xabier)|||/items/2a000d9c-cb5c-4734-a32c-4fef79998c86
Odero, M.D. (Maria Dolores)|||/items/6679bbec-f7dd-480c-a44c-44d7c8493251
McDonald, E.J. (Emma Jane)|||/items/ec0504d2-270e-400a-b9e1-29ca0786f4ad
Killick, S. (Sally)|||/items/c07eb5b9-2a68-430e-bb08-d9d303598e92
Perez, C. (Cristina)|||/items/290e09cd-b084-4cdd-b63e-84762b2d923f
Kondo, T. (Toshinori)|||/items/c7a125af-b58d-42f7-8914-38a5f3062e84
Larrayoz-Ilundáin, M.J. (María José)|||/items/7e29cbb9-798d-4830-bfcf-729a99e05d02
Davies, C. (Carwyn)|||/items/c1de2fdf-b499-47ae-a9e1-a21ffd0bb595
Pellagatti, A. (Andrea)|||/items/a8dd726b-6248-4850-9885-ea609811ceb9
Yip, B.H. (Bon Ham)|||/items/33607d98-632a-4d9b-8863-a9380a676ebb
Fernandez-Mercado, M. (Marta)|||/items/9919a3c8-2c8c-4d98-9b5b-57864191c2bc
author2_role author
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author
author
author
author
author
author
author
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dc.contributor.none.fl_str_mv Dadun. Depósito Académico Digital Universidad de Navarra
dc.subject.none.fl_str_mv Mutation
Acute Myeloid Leukemia
Normal Cytogenetics
topic Mutation
Acute Myeloid Leukemia
Normal Cytogenetics
description Abstract Acute myeloid leukemia patients with normal cytogenetics (CN-AML) account for almost half of AML cases. We aimed to study the frequency and relationship of a wide range of genes previously reported as mutated in AML (ASXL1, NPM1, FLT3, TET2, IDH1/2, RUNX1, DNMT3A, NRAS, JAK2, WT1, CBL, SF3B1, TP53, KRAS and MPL) in a series of 84 CN-AML cases. The most frequently mutated genes in primary cases were NPM1 (60.8%) and FLT3 (50.0%), and in secondary cases ASXL1 (48.5%) and TET2 (30.3%). We showed that 85% of CN-AML patients have mutations in at least one of ASXL1, NPM1, FLT3, TET2, IDH1/2 and/or RUNX1. Serial samples from 19 MDS/CMML cases that progressed to AML were analyzed for ASXL1/TET2/IDH1/2 mutations; seventeen cases presented mutations of at least one of these genes. However, there was no consistent pattern in mutation acquisition during disease progression. This report concerns the analysis of the largest number of gene mutations in CN-AML studied to date, and provides insight into the mutational profile of CN-AML
publishDate 2012
dc.date.none.fl_str_mv 2012
2012-01-01
2012
2012-01-01
2016
2016-08-12
dc.type.none.fl_str_mv journal article
http://purl.org/coar/resource_type/c_6501
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://hdl.handle.net/10171/41454
url https://hdl.handle.net/10171/41454
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
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instname:Universidad de Navarra
instname_str Universidad de Navarra
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