Endothelial Senescence and the Chronic Vascular Diseases: Challenges and Therapeutic Opportunities in Atherosclerosis

Atherosclerosis is probably one of the paradigms of disease linked to aging. Underlying the physiopathology of atherosclerosis are cellular senescence, oxidative stress, and inflammation. These factors are increased in the elderly and from chronic disease patients. Elevated levels of oxidative stres...

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Detalles Bibliográficos
Autores: Alique Aguilar, Matilde|||0000-0002-7912-1133, Ceprían, Noemí, Ramírez Chamond, Manuel Rafael|||0000-0003-4598-339X, Figuer Rubio, Andrea|||0000-0001-9788-0136, Valera, Gemma, Bodega Magro, Guillermo|||0000-0003-1469-3242
Tipo de recurso: artículo
Fecha de publicación:2022
País:España
Institución:Universidad de Alcalá (UAH)
Repositorio:e_Buah Biblioteca Digital Universidad de Alcalá
Idioma:inglés
OAI Identifier:oai:ebuah.uah.es:10017/51249
Acceso en línea:http://hdl.handle.net/10017/51249
https://dx.doi.org/10.3390/jpm12020215
Access Level:acceso abierto
Palabra clave:Atherosclerosis
Cellular senescence
Endothelial senescence
Extracellular vesicles
Inflammation
Senolityc
Fisiología
Physiology
Descripción
Sumario:Atherosclerosis is probably one of the paradigms of disease linked to aging. Underlying the physiopathology of atherosclerosis are cellular senescence, oxidative stress, and inflammation. These factors are increased in the elderly and from chronic disease patients. Elevated levels of oxidative stress affect cellular function and metabolism, inducing senescence. This senescence modifies the cell phenotype into a senescent secretory phenotype. This phenotype activates immune cells, leading to chronic systemic inflammation. Moreover, due to their secretory phenotype, senescence cells present an increased release of highlighted extracellular vesicles that will change nearby/neighborhood cells and paracrine signaling. For this reason, searching for specific senescent cell biomarkers and therapies against the development/killing of senescent cells has become relevant. Recently, senomorphic and senolityc drugs have become relevant in slowing down or eliminating senescence cells. However, even though they have shown promising results in experimental studies, their clinical use is still yet to be determined