IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms

CD40, BLK and BANK1 genes involved in the development and signaling of B-cells are identified as susceptibility loci for numerous inflammatory diseases. Accordingly, we assessed the potential influence of CD40, BLK and BANK1 on the pathogenesis of immunoglobulin-A vasculitis (IgAV), predominantly a...

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Autores: Batista-Liz, Joao Carlos, Genre, Fernanda, Pulito-Cueto, Verónica, Remuzgo-Martínez, Sara, Prieto-Peña, Diana, Márquez, Ana, Ortego-Centeno, Norberto, Leonardo, María Teresa, Peñalba, Ana, Narváez, Javier, Martín-Penagos, Luis, Belmar-Vega, Lara, Gómez Fernández, Cristina, Miranda-Filloy, J. A., Caminal-Montero, Luis, Collado, Paz, Árgila, Diego de, Quiroga, Patricia, Vicente, Esther, Triguero-Martínez, Ana, Rubio, Esteban, León Luque, Manuel, Blanco-Madrigal, Juan María, Galíndez-Agirregoikoa, E., Martín, Javier, Gualillo, Oreste, Blanco, Ricardo, Castañeda, Santos, González-Gay, Miguel A., López-Mejías, Raquel
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2022
País:España
Institución:Consejo Superior de Investigaciones Científicas (CSIC)
Repositorio:DIGITAL.CSIC. Repositorio Institucional del CSIC
OAI Identifier:oai:digital.csic.es:10261/280389
Acceso en línea:http://hdl.handle.net/10261/280389
Access Level:acceso abierto
Palabra clave:IgA vasculitis
Henoch–Schönlein purpura
CD40
BLK
Polymorphisms
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dc.title.none.fl_str_mv IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms
title IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms
spellingShingle IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms
Batista-Liz, Joao Carlos
IgA vasculitis
Henoch–Schönlein purpura
CD40
BLK
Polymorphisms
title_short IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms
title_full IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms
title_fullStr IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms
title_full_unstemmed IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms
title_sort IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms
dc.creator.none.fl_str_mv Batista-Liz, Joao Carlos
Genre, Fernanda
Pulito-Cueto, Verónica
Remuzgo-Martínez, Sara
Prieto-Peña, Diana
Márquez, Ana
Ortego-Centeno, Norberto
Leonardo, María Teresa
Peñalba, Ana
Narváez, Javier
Martín-Penagos, Luis
Belmar-Vega, Lara
Gómez Fernández, Cristina
Miranda-Filloy, J. A.
Caminal-Montero, Luis
Collado, Paz
Árgila, Diego de
Quiroga, Patricia
Vicente, Esther
Triguero-Martínez, Ana
Rubio, Esteban
León Luque, Manuel
Blanco-Madrigal, Juan María
Galíndez-Agirregoikoa, E.
Martín, Javier
Gualillo, Oreste
Blanco, Ricardo
Castañeda, Santos
González-Gay, Miguel A.
López-Mejías, Raquel
author Batista-Liz, Joao Carlos
author_facet Batista-Liz, Joao Carlos
Genre, Fernanda
Pulito-Cueto, Verónica
Remuzgo-Martínez, Sara
Prieto-Peña, Diana
Márquez, Ana
Ortego-Centeno, Norberto
Leonardo, María Teresa
Peñalba, Ana
Narváez, Javier
Martín-Penagos, Luis
Belmar-Vega, Lara
Gómez Fernández, Cristina
Miranda-Filloy, J. A.
Caminal-Montero, Luis
Collado, Paz
Árgila, Diego de
Quiroga, Patricia
Vicente, Esther
Triguero-Martínez, Ana
Rubio, Esteban
León Luque, Manuel
Blanco-Madrigal, Juan María
Galíndez-Agirregoikoa, E.
Martín, Javier
Gualillo, Oreste
Blanco, Ricardo
Castañeda, Santos
González-Gay, Miguel A.
López-Mejías, Raquel
author_role author
author2 Genre, Fernanda
Pulito-Cueto, Verónica
Remuzgo-Martínez, Sara
Prieto-Peña, Diana
Márquez, Ana
Ortego-Centeno, Norberto
Leonardo, María Teresa
Peñalba, Ana
Narváez, Javier
Martín-Penagos, Luis
Belmar-Vega, Lara
Gómez Fernández, Cristina
Miranda-Filloy, J. A.
Caminal-Montero, Luis
Collado, Paz
Árgila, Diego de
Quiroga, Patricia
Vicente, Esther
Triguero-Martínez, Ana
Rubio, Esteban
León Luque, Manuel
Blanco-Madrigal, Juan María
Galíndez-Agirregoikoa, E.
Martín, Javier
Gualillo, Oreste
Blanco, Ricardo
Castañeda, Santos
González-Gay, Miguel A.
López-Mejías, Raquel
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv European Commission
Instituto de Salud Carlos III
Xunta de Galicia
Consejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]
dc.subject.none.fl_str_mv IgA vasculitis
Henoch–Schönlein purpura
CD40
BLK
Polymorphisms
topic IgA vasculitis
Henoch–Schönlein purpura
CD40
BLK
Polymorphisms
description CD40, BLK and BANK1 genes involved in the development and signaling of B-cells are identified as susceptibility loci for numerous inflammatory diseases. Accordingly, we assessed the potential influence of CD40, BLK and BANK1 on the pathogenesis of immunoglobulin-A vasculitis (IgAV), predominantly a B-lymphocyte inflammatory condition. Three genetic variants within CD40 (rs1883832, rs1535045, rs4813003) and BLK (rs2254546, rs2736340, rs2618476) as well as two BANK1 polymorphisms (rs10516487, rs3733197), previously associated with inflammatory diseases, were genotyped in 382 Caucasian patients with IgAV and 955 sex- and ethnically matched healthy controls. No statistically significant differences were observed in the genotype and allele frequencies of CD40, BLK and BANK1 when IgAV patients and healthy controls were compared. Similar results were found when CD40, BLK and BANK1 genotypes or alleles frequencies were compared between patients with IgAV stratified according to the age at disease onset or to the presence/absence of gastrointestinal or renal manifestations. Moreover, no CD40, BLK and BANK1 haplotype differences were disclosed between patients with IgAV and healthy controls and between patients with IgAV stratified according to the clinical characteristics mentioned above. Our findings indicate that CD40, BLK and BANK1 do not contribute to the genetic background of IgAV.
publishDate 2022
dc.date.none.fl_str_mv 2022
2022
2022
2022
dc.type.none.fl_str_mv info:eu-repo/semantics/article
http://purl.org/coar/resource_type/c_6501
Publisher's version
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/10261/280389
url http://hdl.handle.net/10261/280389
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
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info:eu-repo/grantAgreement/ISCIII/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020 (ISCIII)/PI21%2F00042
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https://doi.org/10.3390/jcm11195577

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dc.publisher.none.fl_str_mv Multidisciplinary Digital Publishing Institute
publisher.none.fl_str_mv Multidisciplinary Digital Publishing Institute
dc.source.none.fl_str_mv reponame:DIGITAL.CSIC. Repositorio Institucional del CSIC
instname:Consejo Superior de Investigaciones Científicas (CSIC)
instname_str Consejo Superior de Investigaciones Científicas (CSIC)
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spelling IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene PolymorphismsBatista-Liz, Joao CarlosGenre, FernandaPulito-Cueto, VerónicaRemuzgo-Martínez, SaraPrieto-Peña, DianaMárquez, AnaOrtego-Centeno, NorbertoLeonardo, María TeresaPeñalba, AnaNarváez, JavierMartín-Penagos, LuisBelmar-Vega, LaraGómez Fernández, CristinaMiranda-Filloy, J. A.Caminal-Montero, LuisCollado, PazÁrgila, Diego deQuiroga, PatriciaVicente, EstherTriguero-Martínez, AnaRubio, EstebanLeón Luque, ManuelBlanco-Madrigal, Juan MaríaGalíndez-Agirregoikoa, E.Martín, JavierGualillo, OresteBlanco, RicardoCastañeda, SantosGonzález-Gay, Miguel A.López-Mejías, RaquelIgA vasculitisHenoch–Schönlein purpuraCD40BLKPolymorphismsCD40, BLK and BANK1 genes involved in the development and signaling of B-cells are identified as susceptibility loci for numerous inflammatory diseases. Accordingly, we assessed the potential influence of CD40, BLK and BANK1 on the pathogenesis of immunoglobulin-A vasculitis (IgAV), predominantly a B-lymphocyte inflammatory condition. Three genetic variants within CD40 (rs1883832, rs1535045, rs4813003) and BLK (rs2254546, rs2736340, rs2618476) as well as two BANK1 polymorphisms (rs10516487, rs3733197), previously associated with inflammatory diseases, were genotyped in 382 Caucasian patients with IgAV and 955 sex- and ethnically matched healthy controls. No statistically significant differences were observed in the genotype and allele frequencies of CD40, BLK and BANK1 when IgAV patients and healthy controls were compared. Similar results were found when CD40, BLK and BANK1 genotypes or alleles frequencies were compared between patients with IgAV stratified according to the age at disease onset or to the presence/absence of gastrointestinal or renal manifestations. Moreover, no CD40, BLK and BANK1 haplotype differences were disclosed between patients with IgAV and healthy controls and between patients with IgAV stratified according to the clinical characteristics mentioned above. Our findings indicate that CD40, BLK and BANK1 do not contribute to the genetic background of IgAV.This study was supported by European Union FEDER funds and “Fondo de Investigaciones Sanitarias” (grants PI18/00042 and PI21/00042) from “Instituto de Salud Carlos III” (ISCIII, Health Ministry, Spain). D.P.-P. is a recipient of a Río Hortega program fellowship from the ISCIII, co-funded by the European Social Fund (ESF, “Investing in your future”) (grant number CM20/00006). F.G. is supported by funds of the RICORS Program from ISCIII, co-funded by the European Union (grant number RD21/0002/0025). V.P.-C. is supported by funds of PI18/00042. S.R.-M. is supported by funds of the RETICS Program (RD16/0012/0009) (ISCIII, co-funded by the European Regional Development Fund (ERDF)). O.G. is a staff member of Xunta de Galicia (Servizo Galego de Saude (SERGAS)) through a research-staff stabilization contract (ISCIII/SERGAS) and his work is funded by ISCIII and the European Union FEDER fund (grant numbers RD16/0012/0014 (RIER) and PI17/00409). He is a beneficiary of project funds from the Research Executive Agency (REA) of the European Union in the framework of MSCA-RISE Action of the H2020 Program, project 734899—Olive-Net. R.L.-M. is a recipient of a Miguel Servet type II program fellowship from the ISCIII, co-funded by ESF (“Investing in your future”) (grant number CPII21/00004).Peer reviewedMultidisciplinary Digital Publishing InstituteEuropean CommissionInstituto de Salud Carlos IIIXunta de GaliciaConsejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]2022202220222022info:eu-repo/semantics/articlehttp://purl.org/coar/resource_type/c_6501Publisher's versioninfo:eu-repo/semantics/publishedVersionapplication/pdfhttp://hdl.handle.net/10261/280389reponame:DIGITAL.CSIC. Repositorio Institucional del CSICinstname:Consejo Superior de Investigaciones Científicas (CSIC)Inglés#PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE#info:eu-repo/grantAgreement/ISCIII/Plan Estatal de Investigación Científica y Técnica y de Innovación 2013-2016 (ISCIII)/PI18%2F00042info:eu-repo/grantAgreement/ISCIII/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020 (ISCIII)/PI21%2F00042info:eu-repo/grantAgreement/MINECO//RD16%2F0012%2F0009info:eu-repo/grantAgreement/MINECO//RD16%2F0012%2F0014info:eu-repo/grantAgreement/ISCIII/Plan Estatal de Investigación Científica y Técnica y de Innovación 2013-2016 (ISCIII)/PI17%2F00409info:eu-repo/grantAgreement/EC/H2020/734899https://doi.org/10.3390/jcm11195577Síinfo:eu-repo/semantics/openAccessoai:digital.csic.es:10261/2803892026-05-22T06:33:51Z
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