Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier

Background The Blood-Brain Barrier (BBB) is a complex and dynamic interface that regulates the exchange of molecules and cells between the blood and the central nervous system. It undergoes structural and functional throughout oxidative stress and inflammation, which may compromise its integrity and...

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Autores: Cazalla Ibáñez, Eduardo, Cuadrado Pastor, Antonio, García Yagüe, Ángel Juan
Tipo de recurso: artículo
Fecha de publicación:2024
País:España
Institución:Universidad Autónoma de Madrid
Repositorio:Biblos-e Archivo. Repositorio Institucional de la UAM
Idioma:inglés
OAI Identifier:oai:repositorio.uam.es:10486/728180
Acceso en línea:https://hdl.handle.net/10486/728180
https://dx.doi.org/10.1186/s12987-024-00599-5
Access Level:acceso abierto
Palabra clave:NRF2
Blood-Brain Barrier
Inflammation
Oxidative stress
Neurological disorders
Tight junction
Phytochemicals
Medicina
Química
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spelling Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain BarrierCazalla Ibáñez, EduardoCuadrado Pastor, AntonioGarcía Yagüe, Ángel JuanNRF2Blood-Brain BarrierInflammationOxidative stressNeurological disordersTight junctionPhytochemicalsMedicinaQuímicaBackground The Blood-Brain Barrier (BBB) is a complex and dynamic interface that regulates the exchange of molecules and cells between the blood and the central nervous system. It undergoes structural and functional throughout oxidative stress and inflammation, which may compromise its integrity and contribute to the pathogenesis of neurodegenerative diseases. Main body Maintaining BBB integrity is of utmost importance in preventing a wide range of neurological disorders. NRF2 is the main transcription factor that regulates cellular redox balance and inflammation-related gene expression. It has also demonstrated a potential role in regulating tight junction integrity and contributing to the inhibition of ECM remodeling, by reducing the expression of several metalloprotease family members involved in maintaining BBB function. Overall, we review current insights on the role of NRF2 in addressing protection against the effects of BBB dysfunction, discuss its involvement in BBB maintenance in different neuropathological diseases, as well as, some of its potential activators that have been used in vitro and in vivo animal models for preventing barrier dysfunction. Conclusions Thus, emerging evidence suggests that upregulation of NRF2 and its target genes could suppress oxidative stress, and neuroinflammation, restore BBB integrity, and increase its protectionThis research was funded by the Spanish Ministry of Economy and Competitiveness (MINECO) (grands PDC2021-121421-I00, PDC2022-1337665-I00, PID2022-141786OB-I00 and PID2019-110061RB-I00). The Autonomous Community of Madrid (grands S2017BMD-3827 and P2022_BMD-7230). EC is holder of an FPU contract of MIU (Ministry of Universities FPU2021, FPU21/02505)Springer NatureDepartamento de BioquímicaFacultad de MedicinaGobierno de EspañaComunidad de Madrid20242024-11-21review articlehttp://purl.org/coar/resource_type/c_dcae04bcAMhttp://purl.org/coar/version/c_ab4af688f83e57aainfo:eu-repo/semantics/articleapplication/pdfhttps://hdl.handle.net/10486/728180https://dx.doi.org/10.1186/s12987-024-00599-539574123reponame:Biblos-e Archivo. Repositorio Institucional de la UAMinstname:Universidad Autónoma de MadridInglésengopen accesshttp://purl.org/coar/access_right/c_abf2Attribution-NonCommercial-NoDerivatives 4.0 Internationalhttp://creativecommons.org/licenses/by-nc-nd/4.0/info:eu-repo/semantics/openAccessoai:repositorio.uam.es:10486/7281802026-06-23T12:46:27Z
dc.title.none.fl_str_mv Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier
title Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier
spellingShingle Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier
Cazalla Ibáñez, Eduardo
NRF2
Blood-Brain Barrier
Inflammation
Oxidative stress
Neurological disorders
Tight junction
Phytochemicals
Medicina
Química
title_short Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier
title_full Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier
title_fullStr Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier
title_full_unstemmed Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier
title_sort Role of the transcription factor NRF2 in maintaining the integrity of the Blood-Brain Barrier
dc.creator.none.fl_str_mv Cazalla Ibáñez, Eduardo
Cuadrado Pastor, Antonio
García Yagüe, Ángel Juan
author Cazalla Ibáñez, Eduardo
author_facet Cazalla Ibáñez, Eduardo
Cuadrado Pastor, Antonio
García Yagüe, Ángel Juan
author_role author
author2 Cuadrado Pastor, Antonio
García Yagüe, Ángel Juan
author2_role author
author
dc.contributor.none.fl_str_mv Departamento de Bioquímica
Facultad de Medicina
Gobierno de España
Comunidad de Madrid
dc.subject.none.fl_str_mv NRF2
Blood-Brain Barrier
Inflammation
Oxidative stress
Neurological disorders
Tight junction
Phytochemicals
Medicina
Química
topic NRF2
Blood-Brain Barrier
Inflammation
Oxidative stress
Neurological disorders
Tight junction
Phytochemicals
Medicina
Química
description Background The Blood-Brain Barrier (BBB) is a complex and dynamic interface that regulates the exchange of molecules and cells between the blood and the central nervous system. It undergoes structural and functional throughout oxidative stress and inflammation, which may compromise its integrity and contribute to the pathogenesis of neurodegenerative diseases. Main body Maintaining BBB integrity is of utmost importance in preventing a wide range of neurological disorders. NRF2 is the main transcription factor that regulates cellular redox balance and inflammation-related gene expression. It has also demonstrated a potential role in regulating tight junction integrity and contributing to the inhibition of ECM remodeling, by reducing the expression of several metalloprotease family members involved in maintaining BBB function. Overall, we review current insights on the role of NRF2 in addressing protection against the effects of BBB dysfunction, discuss its involvement in BBB maintenance in different neuropathological diseases, as well as, some of its potential activators that have been used in vitro and in vivo animal models for preventing barrier dysfunction. Conclusions Thus, emerging evidence suggests that upregulation of NRF2 and its target genes could suppress oxidative stress, and neuroinflammation, restore BBB integrity, and increase its protection
publishDate 2024
dc.date.none.fl_str_mv 2024
2024-11-21
dc.type.none.fl_str_mv review article
http://purl.org/coar/resource_type/c_dcae04bc
AM
http://purl.org/coar/version/c_ab4af688f83e57aa
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://hdl.handle.net/10486/728180
https://dx.doi.org/10.1186/s12987-024-00599-5
39574123
url https://hdl.handle.net/10486/728180
https://dx.doi.org/10.1186/s12987-024-00599-5
identifier_str_mv 39574123
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution-NonCommercial-NoDerivatives 4.0 International
http://creativecommons.org/licenses/by-nc-nd/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution-NonCommercial-NoDerivatives 4.0 International
http://creativecommons.org/licenses/by-nc-nd/4.0/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv Springer Nature
publisher.none.fl_str_mv Springer Nature
dc.source.none.fl_str_mv reponame:Biblos-e Archivo. Repositorio Institucional de la UAM
instname:Universidad Autónoma de Madrid
instname_str Universidad Autónoma de Madrid
reponame_str Biblos-e Archivo. Repositorio Institucional de la UAM
collection Biblos-e Archivo. Repositorio Institucional de la UAM
repository.name.fl_str_mv
repository.mail.fl_str_mv
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