Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling

Small and large preclinical animal models have shown that antagomir-92a-based therapy reduces early postischemic loss of function, but its effect on postinfarction remodeling is not known. In addition, the reported remote miR-92a inhibition in noncardiac organs prevents the translation of nonvectori...

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Autores: Bellera Gotarda, Neus|||0000-0002-1040-0900, Barba, Ignasi|||0000-0001-9886-3776, Rodríguez Sinovas, Antonio|||0000-0003-2930-8773, Ferret, Eulalia, Asín, Miguel Angel, Gonzalez-Alujas, MªTeresa, Pérez-Rodón, Jordi|||0000-0003-2234-5248, Esteves, Marielle, Fonseca, Carla, Toran, Nuria, García del Blanco, Bruno|||0000-0002-4527-1600, Pérez, Amadeo, García-Dorado, David|||0000-0002-1126-1279
Tipo de recurso: artículo
Fecha de publicación:2014
País:España
Institución:Universitat Autònoma de Barcelona
Repositorio:Dipòsit Digital de Documents de la UAB
Idioma:inglés
OAI Identifier:oai:ddd.uab.cat:185098
Acceso en línea:https://ddd.uab.cat/record/185098
https://dx.doi.org/urn:doi:10.1161/JAHA.114.000946
Access Level:acceso abierto
Palabra clave:Angiogenesis
Antagomir
Heart failure
MicroRNA
Myocardial infarction
Remodeling
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spelling Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct RemodelingBellera Gotarda, Neus|||0000-0002-1040-0900Barba, Ignasi|||0000-0001-9886-3776Rodríguez Sinovas, Antonio|||0000-0003-2930-8773Ferret, EulaliaAsín, Miguel AngelGonzalez-Alujas, MªTeresaPérez-Rodón, Jordi|||0000-0003-2234-5248Esteves, MarielleFonseca, CarlaToran, NuriaGarcía del Blanco, Bruno|||0000-0002-4527-1600Pérez, AmadeoGarcía-Dorado, David|||0000-0002-1126-1279AngiogenesisAntagomirHeart failureMicroRNAMyocardial infarctionRemodelingSmall and large preclinical animal models have shown that antagomir-92a-based therapy reduces early postischemic loss of function, but its effect on postinfarction remodeling is not known. In addition, the reported remote miR-92a inhibition in noncardiac organs prevents the translation of nonvectorized miR-targeted therapy to the clinical setting. We investigated whether a single intracoronary administration of antagomir-92a encapsulated in microspheres could prevent deleterious remodeling of myocardium 1 month after acute myocardial infarction AUTHOR: Should "acute" be added before "myocardial infarction" (since abbreviation is AMI)? Also check at first mention in main text (AMI) without adverse effects. In a percutaneous pig model of reperfused AMI, a single intracoronary administration of antagomir-92a encapsulated in specific microspheres (9 μm poly-d,-lactide-co-glycolide [PLGA]) inhibited miR-92a in a local, selective, and sustained manner (n=3 pigs euthanized 1, 3, and 10 days after treatment; 8×, 2×, and 5×-fold inhibition at 1, 3, and 10 days). Downregulation of miR-92a resulted in significant vessel growth (n=27 adult minipigs randomly allocated to blind receive encapsulated antagomir-92a, encapsulated placebo, or saline [n=8, 9, 9]; P =0.001), reduced regional wall-motion dysfunction (P =0.03), and prevented adverse remodeling in the infarct area 1 month after injury (P =0.03). Intracoronary injection of microspheres had no significant adverse effect in downstream myocardium in healthy pigs (n=2), and fluorescein isothiocyanate albumin-PLGA microspheres were not found in myocardium outside the left anterior descending coronary artery territory (n=4) or in other organs (n=2). Early single intracoronary administration of encapsulated antagomir-92a in an adult pig model of reperfused AMI prevents left ventricular remodeling with no local or distant adverse effects, emerging as a promising therapeutic approach to translate to patients who suffer a large AMI.Universitat Autònoma de Barcelona 22014-01-0120142014-01-01Articlehttp://purl.org/coar/resource_type/c_6501VoRhttp://purl.org/coar/version/c_970fb48d4fbd8a85info:eu-repo/semantics/articleapplication/pdfhttps://ddd.uab.cat/record/185098https://dx.doi.org/urn:doi:10.1161/JAHA.114.000946reponame:Dipòsit Digital de Documents de la UABinstname:Universitat Autònoma de BarcelonaInglésengMinisterio de Ciencia e Innovación https://doi.org/10.13039/501100004837 SAF2008-03067Instituto de Salud Carlos III https://doi.org/10.13039/501100004587 RD12-0042-0021open accesshttp://purl.org/coar/access_right/c_abf2Aquest document està subjecte a una llicència d'ús Creative Commons. Es permet la reproducció total o parcial, la distribució, la comunicació pública de l'obra i la creació d'obres derivades, sempre que no sigui amb finalitats comercials, i sempre que es reconegui l'autoria de l'obra original.https://creativecommons.org/licenses/by-nc/4.0/info:eu-repo/semantics/openAccessoai:ddd.uab.cat:1850982026-06-06T12:50:31Z
dc.title.none.fl_str_mv Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling
title Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling
spellingShingle Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling
Bellera Gotarda, Neus|||0000-0002-1040-0900
Angiogenesis
Antagomir
Heart failure
MicroRNA
Myocardial infarction
Remodeling
title_short Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling
title_full Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling
title_fullStr Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling
title_full_unstemmed Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling
title_sort Single Intracoronary Injection of Encapsulated Antagomir-92a Promotes Angiogenesis and Prevents Adverse Infarct Remodeling
dc.creator.none.fl_str_mv Bellera Gotarda, Neus|||0000-0002-1040-0900
Barba, Ignasi|||0000-0001-9886-3776
Rodríguez Sinovas, Antonio|||0000-0003-2930-8773
Ferret, Eulalia
Asín, Miguel Angel
Gonzalez-Alujas, MªTeresa
Pérez-Rodón, Jordi|||0000-0003-2234-5248
Esteves, Marielle
Fonseca, Carla
Toran, Nuria
García del Blanco, Bruno|||0000-0002-4527-1600
Pérez, Amadeo
García-Dorado, David|||0000-0002-1126-1279
author Bellera Gotarda, Neus|||0000-0002-1040-0900
author_facet Bellera Gotarda, Neus|||0000-0002-1040-0900
Barba, Ignasi|||0000-0001-9886-3776
Rodríguez Sinovas, Antonio|||0000-0003-2930-8773
Ferret, Eulalia
Asín, Miguel Angel
Gonzalez-Alujas, MªTeresa
Pérez-Rodón, Jordi|||0000-0003-2234-5248
Esteves, Marielle
Fonseca, Carla
Toran, Nuria
García del Blanco, Bruno|||0000-0002-4527-1600
Pérez, Amadeo
García-Dorado, David|||0000-0002-1126-1279
author_role author
author2 Barba, Ignasi|||0000-0001-9886-3776
Rodríguez Sinovas, Antonio|||0000-0003-2930-8773
Ferret, Eulalia
Asín, Miguel Angel
Gonzalez-Alujas, MªTeresa
Pérez-Rodón, Jordi|||0000-0003-2234-5248
Esteves, Marielle
Fonseca, Carla
Toran, Nuria
García del Blanco, Bruno|||0000-0002-4527-1600
Pérez, Amadeo
García-Dorado, David|||0000-0002-1126-1279
author2_role author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Universitat Autònoma de Barcelona
dc.subject.none.fl_str_mv Angiogenesis
Antagomir
Heart failure
MicroRNA
Myocardial infarction
Remodeling
topic Angiogenesis
Antagomir
Heart failure
MicroRNA
Myocardial infarction
Remodeling
description Small and large preclinical animal models have shown that antagomir-92a-based therapy reduces early postischemic loss of function, but its effect on postinfarction remodeling is not known. In addition, the reported remote miR-92a inhibition in noncardiac organs prevents the translation of nonvectorized miR-targeted therapy to the clinical setting. We investigated whether a single intracoronary administration of antagomir-92a encapsulated in microspheres could prevent deleterious remodeling of myocardium 1 month after acute myocardial infarction AUTHOR: Should "acute" be added before "myocardial infarction" (since abbreviation is AMI)? Also check at first mention in main text (AMI) without adverse effects. In a percutaneous pig model of reperfused AMI, a single intracoronary administration of antagomir-92a encapsulated in specific microspheres (9 μm poly-d,-lactide-co-glycolide [PLGA]) inhibited miR-92a in a local, selective, and sustained manner (n=3 pigs euthanized 1, 3, and 10 days after treatment; 8×, 2×, and 5×-fold inhibition at 1, 3, and 10 days). Downregulation of miR-92a resulted in significant vessel growth (n=27 adult minipigs randomly allocated to blind receive encapsulated antagomir-92a, encapsulated placebo, or saline [n=8, 9, 9]; P =0.001), reduced regional wall-motion dysfunction (P =0.03), and prevented adverse remodeling in the infarct area 1 month after injury (P =0.03). Intracoronary injection of microspheres had no significant adverse effect in downstream myocardium in healthy pigs (n=2), and fluorescein isothiocyanate albumin-PLGA microspheres were not found in myocardium outside the left anterior descending coronary artery territory (n=4) or in other organs (n=2). Early single intracoronary administration of encapsulated antagomir-92a in an adult pig model of reperfused AMI prevents left ventricular remodeling with no local or distant adverse effects, emerging as a promising therapeutic approach to translate to patients who suffer a large AMI.
publishDate 2014
dc.date.none.fl_str_mv 2
2014-01-01
2014
2014-01-01
dc.type.none.fl_str_mv Article
http://purl.org/coar/resource_type/c_6501
VoR
http://purl.org/coar/version/c_970fb48d4fbd8a85
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://ddd.uab.cat/record/185098
https://dx.doi.org/urn:doi:10.1161/JAHA.114.000946
url https://ddd.uab.cat/record/185098
https://dx.doi.org/urn:doi:10.1161/JAHA.114.000946
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.relation.none.fl_str_mv Ministerio de Ciencia e Innovación https://doi.org/10.13039/501100004837 SAF2008-03067
Instituto de Salud Carlos III https://doi.org/10.13039/501100004587 RD12-0042-0021
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by-nc/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by-nc/4.0/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.source.none.fl_str_mv reponame:Dipòsit Digital de Documents de la UAB
instname:Universitat Autònoma de Barcelona
instname_str Universitat Autònoma de Barcelona
reponame_str Dipòsit Digital de Documents de la UAB
collection Dipòsit Digital de Documents de la UAB
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