Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes

Background: Telomeres are nucleoprotein structures present at the terminal region of the chromosomes. Mutations in genes coding for proteins involved in telomere maintenance are causative of a number of disorders known as telomeropathies. The genetic origin of these diseases is heterogeneous and has...

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Autores: García Arias-Salgado, Elena, Gálvez, Eva, Planas-Cerezales, Lurdes|||0000-0001-7636-444X, Pintado-Berninches, Laura|||0000-0002-3331-7130, Vallespín, Elena|||0000-0002-8080-8629, Martínez, Pilar, Carrillo García, Jaime|||0000-0002-2861-3015, Iarriccio, Laura, Ruiz-Llobet, Anna|||0000-0002-0155-7922, Català, Albert|||0000-0003-0133-1752, Badell Serra, Isabel|||0000-0001-6546-2175, Gonzalez-Granado, Luis Ignacio|||0000-0001-6917-8980, Martín-Nalda, Andrea|||0000-0002-1715-153X, Martínez Gallo, Mónica|||0000-0002-7340-2161, Galera, Ana, Rodríguez-Vigil, Carmen, Bastos, Mariana|||0000-0002-9431-4646, Pérez de Nanclares, Guiomar|||0000-0002-2424-5294, Leiro-Fernández, Virginia, Uria Oficialdegui, Maria Luz|||0000-0002-1384-999X, Díaz de Heredia, Cristina|||0000-0001-8086-296X, Valenzuela, Claudia|||0000-0002-0856-6290, Martín Alegre, Sara|||0000-0002-0619-9255, López-Muñiz, Belén, Lapunzina, Pablo|||0000-0002-6324-4825, Sevilla, Julián|||0000-0002-6852-1860, Molina Molina, Maria|||0000-0002-1852-1723, Perona, Rosario|||0000-0002-2973-5153, Sastre, Leandro|||0000-0003-3613-5938
Tipo de recurso: artículo
Fecha de publicación:2019
País:España
Institución:Universitat Autònoma de Barcelona
Repositorio:Dipòsit Digital de Documents de la UAB
Idioma:inglés
OAI Identifier:oai:ddd.uab.cat:223237
Acceso en línea:https://ddd.uab.cat/record/223237
https://dx.doi.org/urn:doi:10.1186/s13023-019-1046-0
Access Level:acceso abierto
Palabra clave:Telomere
Dyskeratosis congenita
Pulmonary fibrosis
Aplastic anemia
Telomeropathies
DNA repair
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spelling Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genesGarcía Arias-Salgado, ElenaGálvez, EvaPlanas-Cerezales, Lurdes|||0000-0001-7636-444XPintado-Berninches, Laura|||0000-0002-3331-7130Vallespín, Elena|||0000-0002-8080-8629Martínez, PilarCarrillo García, Jaime|||0000-0002-2861-3015Iarriccio, LauraRuiz-Llobet, Anna|||0000-0002-0155-7922Català, Albert|||0000-0003-0133-1752Badell Serra, Isabel|||0000-0001-6546-2175Gonzalez-Granado, Luis Ignacio|||0000-0001-6917-8980Martín-Nalda, Andrea|||0000-0002-1715-153XMartínez Gallo, Mónica|||0000-0002-7340-2161Galera, AnaRodríguez-Vigil, CarmenBastos, Mariana|||0000-0002-9431-4646Pérez de Nanclares, Guiomar|||0000-0002-2424-5294Leiro-Fernández, VirginiaUria Oficialdegui, Maria Luz|||0000-0002-1384-999XDíaz de Heredia, Cristina|||0000-0001-8086-296XValenzuela, Claudia|||0000-0002-0856-6290Martín Alegre, Sara|||0000-0002-0619-9255López-Muñiz, BelénLapunzina, Pablo|||0000-0002-6324-4825Sevilla, Julián|||0000-0002-6852-1860Molina Molina, Maria|||0000-0002-1852-1723Perona, Rosario|||0000-0002-2973-5153Sastre, Leandro|||0000-0003-3613-5938TelomereDyskeratosis congenitaPulmonary fibrosisAplastic anemiaTelomeropathiesDNA repairBackground: Telomeres are nucleoprotein structures present at the terminal region of the chromosomes. Mutations in genes coding for proteins involved in telomere maintenance are causative of a number of disorders known as telomeropathies. The genetic origin of these diseases is heterogeneous and has not been determined for a significant proportion of patients. Methods: This article describes the genetic characterization of a cohort of patients. Telomere length was determined by Southern blot and quantitative PCR. Nucleotide variants were analyzed either by high-resolution melting analysis and Sanger sequencing of selected exons or by massive sequencing of a panel of genes. Results: Forty-seven patients with telomere length below the 10% of normal population, affected with three telomeropathies: dyskeratosis congenita (4), aplastic anemia (22) or pulmonary fibrosis (21) were analyzed. Eighteen of these patients presented known pathogenic or novel possibly pathogenic variants in the telomere-related genes TERT, TERC, RTEL1, CTC1 and ACD. In addition, the analyses of a panel of 188 genes related to haematological disorders indicated that a relevant proportion of the patients (up to 35%) presented rare variants in genes related to DNA repair or in genes coding for proteins involved in the resolution of complex DNA structures, that participate in telomere replication. Mutations in some of these genes are causative of several syndromes previously associated to telomere shortening. Conclusion: Novel variants in telomere, DNA repair and replication genes are described that might indicate the contribution of variants in these genes to the development of telomeropathies. Patients carrying variants in telomere-related genes presented worse evolution after diagnosis than the rest of patients analyzed. 22019-01-0120192019-01-01Articlehttp://purl.org/coar/resource_type/c_6501VoRhttp://purl.org/coar/version/c_970fb48d4fbd8a85info:eu-repo/semantics/articleapplication/pdfhttps://ddd.uab.cat/record/223237https://dx.doi.org/urn:doi:10.1186/s13023-019-1046-0reponame:Dipòsit Digital de Documents de la UABinstname:Universitat Autònoma de BarcelonaInglésengInstituto de Salud Carlos III https://doi.org/10.13039/501100004587 PI17-01401Instituto de Salud Carlos III https://doi.org/10.13039/501100004587 PI14-01495open accesshttp://purl.org/coar/access_right/c_abf2Aquest document està subjecte a una llicència d'ús Creative Commons. Es permet la reproducció total o parcial, la distribució, la comunicació pública de l'obra i la creació d'obres derivades, fins i tot amb finalitats comercials, sempre i quan es reconegui l'autoria de l'obra original.https://creativecommons.org/licenses/by/4.0/info:eu-repo/semantics/openAccessoai:ddd.uab.cat:2232372026-06-06T12:50:31Z
dc.title.none.fl_str_mv Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes
title Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes
spellingShingle Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes
García Arias-Salgado, Elena
Telomere
Dyskeratosis congenita
Pulmonary fibrosis
Aplastic anemia
Telomeropathies
DNA repair
title_short Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes
title_full Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes
title_fullStr Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes
title_full_unstemmed Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes
title_sort Genetic analyses of aplastic anemia and idiopathic pulmonary fibrosis patients with short telomeres, possible implication of DNA-repair genes
dc.creator.none.fl_str_mv García Arias-Salgado, Elena
Gálvez, Eva
Planas-Cerezales, Lurdes|||0000-0001-7636-444X
Pintado-Berninches, Laura|||0000-0002-3331-7130
Vallespín, Elena|||0000-0002-8080-8629
Martínez, Pilar
Carrillo García, Jaime|||0000-0002-2861-3015
Iarriccio, Laura
Ruiz-Llobet, Anna|||0000-0002-0155-7922
Català, Albert|||0000-0003-0133-1752
Badell Serra, Isabel|||0000-0001-6546-2175
Gonzalez-Granado, Luis Ignacio|||0000-0001-6917-8980
Martín-Nalda, Andrea|||0000-0002-1715-153X
Martínez Gallo, Mónica|||0000-0002-7340-2161
Galera, Ana
Rodríguez-Vigil, Carmen
Bastos, Mariana|||0000-0002-9431-4646
Pérez de Nanclares, Guiomar|||0000-0002-2424-5294
Leiro-Fernández, Virginia
Uria Oficialdegui, Maria Luz|||0000-0002-1384-999X
Díaz de Heredia, Cristina|||0000-0001-8086-296X
Valenzuela, Claudia|||0000-0002-0856-6290
Martín Alegre, Sara|||0000-0002-0619-9255
López-Muñiz, Belén
Lapunzina, Pablo|||0000-0002-6324-4825
Sevilla, Julián|||0000-0002-6852-1860
Molina Molina, Maria|||0000-0002-1852-1723
Perona, Rosario|||0000-0002-2973-5153
Sastre, Leandro|||0000-0003-3613-5938
author García Arias-Salgado, Elena
author_facet García Arias-Salgado, Elena
Gálvez, Eva
Planas-Cerezales, Lurdes|||0000-0001-7636-444X
Pintado-Berninches, Laura|||0000-0002-3331-7130
Vallespín, Elena|||0000-0002-8080-8629
Martínez, Pilar
Carrillo García, Jaime|||0000-0002-2861-3015
Iarriccio, Laura
Ruiz-Llobet, Anna|||0000-0002-0155-7922
Català, Albert|||0000-0003-0133-1752
Badell Serra, Isabel|||0000-0001-6546-2175
Gonzalez-Granado, Luis Ignacio|||0000-0001-6917-8980
Martín-Nalda, Andrea|||0000-0002-1715-153X
Martínez Gallo, Mónica|||0000-0002-7340-2161
Galera, Ana
Rodríguez-Vigil, Carmen
Bastos, Mariana|||0000-0002-9431-4646
Pérez de Nanclares, Guiomar|||0000-0002-2424-5294
Leiro-Fernández, Virginia
Uria Oficialdegui, Maria Luz|||0000-0002-1384-999X
Díaz de Heredia, Cristina|||0000-0001-8086-296X
Valenzuela, Claudia|||0000-0002-0856-6290
Martín Alegre, Sara|||0000-0002-0619-9255
López-Muñiz, Belén
Lapunzina, Pablo|||0000-0002-6324-4825
Sevilla, Julián|||0000-0002-6852-1860
Molina Molina, Maria|||0000-0002-1852-1723
Perona, Rosario|||0000-0002-2973-5153
Sastre, Leandro|||0000-0003-3613-5938
author_role author
author2 Gálvez, Eva
Planas-Cerezales, Lurdes|||0000-0001-7636-444X
Pintado-Berninches, Laura|||0000-0002-3331-7130
Vallespín, Elena|||0000-0002-8080-8629
Martínez, Pilar
Carrillo García, Jaime|||0000-0002-2861-3015
Iarriccio, Laura
Ruiz-Llobet, Anna|||0000-0002-0155-7922
Català, Albert|||0000-0003-0133-1752
Badell Serra, Isabel|||0000-0001-6546-2175
Gonzalez-Granado, Luis Ignacio|||0000-0001-6917-8980
Martín-Nalda, Andrea|||0000-0002-1715-153X
Martínez Gallo, Mónica|||0000-0002-7340-2161
Galera, Ana
Rodríguez-Vigil, Carmen
Bastos, Mariana|||0000-0002-9431-4646
Pérez de Nanclares, Guiomar|||0000-0002-2424-5294
Leiro-Fernández, Virginia
Uria Oficialdegui, Maria Luz|||0000-0002-1384-999X
Díaz de Heredia, Cristina|||0000-0001-8086-296X
Valenzuela, Claudia|||0000-0002-0856-6290
Martín Alegre, Sara|||0000-0002-0619-9255
López-Muñiz, Belén
Lapunzina, Pablo|||0000-0002-6324-4825
Sevilla, Julián|||0000-0002-6852-1860
Molina Molina, Maria|||0000-0002-1852-1723
Perona, Rosario|||0000-0002-2973-5153
Sastre, Leandro|||0000-0003-3613-5938
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.subject.none.fl_str_mv Telomere
Dyskeratosis congenita
Pulmonary fibrosis
Aplastic anemia
Telomeropathies
DNA repair
topic Telomere
Dyskeratosis congenita
Pulmonary fibrosis
Aplastic anemia
Telomeropathies
DNA repair
description Background: Telomeres are nucleoprotein structures present at the terminal region of the chromosomes. Mutations in genes coding for proteins involved in telomere maintenance are causative of a number of disorders known as telomeropathies. The genetic origin of these diseases is heterogeneous and has not been determined for a significant proportion of patients. Methods: This article describes the genetic characterization of a cohort of patients. Telomere length was determined by Southern blot and quantitative PCR. Nucleotide variants were analyzed either by high-resolution melting analysis and Sanger sequencing of selected exons or by massive sequencing of a panel of genes. Results: Forty-seven patients with telomere length below the 10% of normal population, affected with three telomeropathies: dyskeratosis congenita (4), aplastic anemia (22) or pulmonary fibrosis (21) were analyzed. Eighteen of these patients presented known pathogenic or novel possibly pathogenic variants in the telomere-related genes TERT, TERC, RTEL1, CTC1 and ACD. In addition, the analyses of a panel of 188 genes related to haematological disorders indicated that a relevant proportion of the patients (up to 35%) presented rare variants in genes related to DNA repair or in genes coding for proteins involved in the resolution of complex DNA structures, that participate in telomere replication. Mutations in some of these genes are causative of several syndromes previously associated to telomere shortening. Conclusion: Novel variants in telomere, DNA repair and replication genes are described that might indicate the contribution of variants in these genes to the development of telomeropathies. Patients carrying variants in telomere-related genes presented worse evolution after diagnosis than the rest of patients analyzed.
publishDate 2019
dc.date.none.fl_str_mv 2
2019-01-01
2019
2019-01-01
dc.type.none.fl_str_mv Article
http://purl.org/coar/resource_type/c_6501
VoR
http://purl.org/coar/version/c_970fb48d4fbd8a85
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://ddd.uab.cat/record/223237
https://dx.doi.org/urn:doi:10.1186/s13023-019-1046-0
url https://ddd.uab.cat/record/223237
https://dx.doi.org/urn:doi:10.1186/s13023-019-1046-0
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.relation.none.fl_str_mv Instituto de Salud Carlos III https://doi.org/10.13039/501100004587 PI17-01401
Instituto de Salud Carlos III https://doi.org/10.13039/501100004587 PI14-01495
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by/4.0/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.source.none.fl_str_mv reponame:Dipòsit Digital de Documents de la UAB
instname:Universitat Autònoma de Barcelona
instname_str Universitat Autònoma de Barcelona
reponame_str Dipòsit Digital de Documents de la UAB
collection Dipòsit Digital de Documents de la UAB
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