Enhanced Susceptibility to Tomato Chlorosis Virus (ToCV) in Hsp90- and Sgt1 -Silenced Plants

The emerging whitefly-transmitted crinivirus tomato chlorosis virus (ToCV) causes substantial economic losses by inducing yellow leaf disorder in tomato crops. This study explores potential resistance mechanisms by examining early-stage molecular responses to ToCV. A time-course transcriptome analys...

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Detalhes bibliográficos
Autores: Ontiveros, Irene|||0000-0003-0090-2330, Fernández-Pozo, Noé|||0000-0002-6489-5566, Esteve-Codina, Anna|||0000-0003-0361-2873, Lopez-Moya, Juan Jose|||0000-0002-3176-9651, Díaz Pendón, Juan Antonio|||0000-0002-3616-6622
Formato: artículo
Fecha de publicación:2023
País:España
Recursos:Universitat Autònoma de Barcelona
Repositorio:Dipòsit Digital de Documents de la UAB
Idioma:inglés
OAI Identifier:oai:ddd.uab.cat:296867
Acesso em linha:https://ddd.uab.cat/record/296867
https://dx.doi.org/urn:doi:10.3390/v15122370
Access Level:acceso abierto
Palavra-chave:Basal resistance
Hsp90
Sgt1
ToCV
Bemisia tabaci
Tomato
Descrição
Resumo:The emerging whitefly-transmitted crinivirus tomato chlorosis virus (ToCV) causes substantial economic losses by inducing yellow leaf disorder in tomato crops. This study explores potential resistance mechanisms by examining early-stage molecular responses to ToCV. A time-course transcriptome analysis compared naïve, mock, and ToCV-infected plants at 2, 7, and 14 days post-infection (dpi). Gene expression changes were most notable at 2 and 14 dpi, likely corresponding to whitefly feeding and viral infection. Gene Ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) enrichment analyses revealed key genes and pathways associated with ToCV infection, including those related to plant immunity, flavonoid and steroid biosynthesis, photosynthesis, and hormone signaling. Additionally, virus-derived small interfering RNAs (vsRNAs) originating from ToCV predominantly came from RNA2 and were 22 nucleotides in length. Furthermore, two genes involved in plant immunity, Hsp90 (heat shock protein 90) and its co-chaperone Sgt1 (suppressor of the G2 allele of Skp1) were targeted through viral-induced gene silencing (VIGS), showing a potential contribution to basal resistance against viral infections since their reduction correlated with increased ToCV accumulation. This study provides insights into tomato plant responses to ToCV, with potential implications for developing effective disease control strategies.