Blood vessels are in control : vasculature regulates the neural niche in cranial sensory ganglia

Cranial sensory ganglia are groups of neurons located in the head of chordates outside the central nervous system that allow individuals to sense and perceive information from the outer world. The regulation of neural proliferation and differentiation is key for cranial ganglia formation and functio...

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Detalles Bibliográficos
Autor: Taberner Pérez, Laura
Tipo de recurso: tesis doctoral
Estado:Versión publicada
Fecha de publicación:2019
País:España
Institución:CBUC, CESCA
Repositorio:TDR. Tesis Doctorales en Red
OAI Identifier:oai:www.tdx.cat:10803/668054
Acceso en línea:http://hdl.handle.net/10803/668054
Access Level:acceso abierto
Palabra clave:Neurovascular
Cranial ganglia
Dll4/Notch
Cytonemes
Bood flow
Ganglis granials
Flux sanguini
616.8
Descripción
Sumario:Cranial sensory ganglia are groups of neurons located in the head of chordates outside the central nervous system that allow individuals to sense and perceive information from the outer world. The regulation of neural proliferation and differentiation is key for cranial ganglia formation and functioning. This control is carried out by signals provided by their niche. Blood vessels have emerged as key components in the adult neural stem cell niche. However, the putative function of vasculature on neural behaviour has yet not been studied in the peripheral nervous system. In the present work, I have described the anatomical relationship of cranial sensory ganglia with vasculature during their development, focusing in the statoacoustic ganglion and using zebrafish as a model system. Secondly, I have demonstrated that two independent signalling mechanisms exist from vasculature to the developing sensory neurons. Early in development, endothelial cells maintain neural cells’ quiescence via Dll4/Notch1 signalling and cytoneme contacts. Later, blood flow onset produces transcriptional changes in neural cells related to oxygen sensing and is required for the differentiation of sensory neurons.