Longitudinal trajectory of plasma p-tau217 in cognitively unimpaired subjects

Background: The advent of Alzheimer's disease-modifying drugs requires accurate biological diagnosis to identify candidates for these therapies. So far, the most promising single plasma biomarker is phosphorylated tau at threonine 217 (p-tau217). To understand its biological features, it is ess...

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Detalhes bibliográficos
Autores: Martínez Dubarbie, Francisco, Guerra Ruiz, Armando Raúl, López García, Sara, Lage Martínez, Carmen, Fernández Matarrubia, Marta, Pozueta Cantudo, Ana, García-Martínez, María, Corrales Pardo, Andrea|||0000-0001-9616-6985, Bravo González, María Paz, López Hoyos, Marcos, Irure Ventura, Juan, Marco de Lucas, Enrique|||0000-0002-5310-7245, Drake Pérez, Marta|||0000-0002-4049-2885, García Unzueta, María Teresa, Sánchez Juan, Pascual, Rodríguez Rodríguez, Eloy Manuel
Tipo de documento: artigo
Data de publicação:2024
País:España
Recursos:Universidad de Cantabria (UC)
Repositório:UCrea Repositorio Abierto de la Universidad de Cantabria
Idioma:inglês
OAI Identifier:oai:repositorio.unican.es:10902/36080
Acesso em linha:https://hdl.handle.net/10902/36080
Access Level:Acceso aberto
Palavra-chave:Plasma p-tau217
Alzheimer’s disease
Early diagnosis
Longitudinal
Healthy controls
Biomarkers
Descrição
Resumo:Background: The advent of Alzheimer's disease-modifying drugs requires accurate biological diagnosis to identify candidates for these therapies. So far, the most promising single plasma biomarker is phosphorylated tau at threonine 217 (p-tau217). To understand its biological features, it is essential to know its longitudinal trajectory and factors influencing it in cognitively unimpaired subjects with no brain pathology. Methods: We analyzed longitudinal plasma p-tau217 values (mean follow-up time = 768.3 days) in a cohort of 209 healthy volunteers. We have studied factors associated with plasma p-tau217 changes by using different linear mixed-effects models. Results: In amyloid-negative cognitively healthy subjects (n = 151) carriers of ApoE epsilon4 allele had significantly higher p-tau217 values than non-carriers (0.85 pg/mL; p-value < 0.001) and also a greater rate of change (0.01 pg/mL/year; p-value < 0.001). In the overall sample, including subjects with amyloid and tau pathology we have seen that amyloid positive subjects had higher predicted baseline plasma p-tau217 values than amyloid negative subjects (0.16 pg/mL; p-value < 0.001) and a greater rate of change (0.00004 pg/mL/day; p-value < 0.001). Subjects considered tau positive also showed a greater rate of change of p-tau217 with respect to tau negative (0.00005 pg/mL/day; p-value < 0.001). A + T + N + participants showed a higher baseline p-tau217 levels than A-T-N- subjects (0.2 pg/mL; p-value < 0.001) and also a greater rate of change (0.00006 pg/mL/day; p-value = 0.002). ApoE epsilon4 carriers had a greater rate of change than non-carriers (0.00003 pg/mL/day; p-value = 0.03). Conclusion: In amyloid-negative cognitively unimpaired subjects, ApoE4 status influenced both baseline levels and rate of change of plasma p-tau217. Other factors such as age, sex or glomerular filtration rate have not shown significant influence on plasma p-tau217 levels in this group.