African swine fever virus infection in Classical swine fever subclinically infected wild boars

Recently moderate-virulence classical swine fever virus (CSFV) strains have been proven capable of generating postnatal persistent infection (PI), defined by the maintenance of viremia and the inability to generate CSFV-specific immune responses in animals. These animals also showed a type I interfe...

Full description

Bibliographic Details
Authors: Cabezón Ponsoda, Óscar, Muñoz González, Sara, Colom-Cadena, Andreu, Rosell, Rosa, Lavín González, Santiago, Marco, Ignasi, Fraile Sauce, Lorenzo José, Martínez de la Riva, Paloma, Rodríguez, Fernando, Domínguez, Javier, Ganges, Llilianne
Format: article
Status:Published version
Publication Date:2017
Country:España
Institution:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
Repository:Recercat. Dipósit de la Recerca de Catalunya
OAI Identifier:oai:recercat.cat:10459.1/60302
Online Access:https://doi.org/10.1186/s12917-017-1150-0
http://hdl.handle.net/10459.1/60302
Access Level:Open access
Keyword:CSFV
CSF postnatal persistent infection
Subclinical CSF
ASFV
Description
Summary:Recently moderate-virulence classical swine fever virus (CSFV) strains have been proven capable of generating postnatal persistent infection (PI), defined by the maintenance of viremia and the inability to generate CSFV-specific immune responses in animals. These animals also showed a type I interferon blockade in the absence of clinical signs. In this study, we assessed the infection generated in 7-week-old CSFV PI wild boars after infection with the African swine fever virus (ASFV). The wild boars were divided in two groups and were infected with ASFV. Group A comprised boars who were CSFV PI in a subclinical form and Group B comprised pestivirus-free wild boars. Some relevant parameters related to CSFV replication and the immune response of CSFV PI animals were studied. Additionally, serum soluble factors such as IFN-α, TNF-α, IL-6, IL-10, IFN-γ and sCD163 were analysed before and after ASFV infection to assess their role in disease progression.