Unusual viral ligand with alternative interactions is presented by HLA-Cw4 in human respiratory syncytial virus-infected cells.

Short viral antigens bound to human major histocompatibility complex (HLA) class I molecules are presented on infected cells. Vaccine development frequently relies on synthetic peptides to identify optimal HLA class I ligands. However, when natural peptides are analyzed, more complex mixtures are fo...

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Detalles Bibliográficos
Autores: Infantes, Susana, Lorente, Elena, Cragnolini, Juan José, Ramos, Manuel, Garcia, Ruth, Jimenez, Mercedes, Iborra, Salvador, Val, Margarita del, Lopez, Daniel
Tipo de recurso: artículo
Fecha de publicación:2011
País:España
Institución:Instituto de Salud Carlos III (ISCIII)
Repositorio:Repisalud
Idioma:inglés
OAI Identifier:oai:repisalud.isciii.es:20.500.12105/10659
Acceso en línea:http://hdl.handle.net/20.500.12105/10659
Access Level:acceso abierto
Palabra clave:Ligands
Animals
Antigens, Viral
Cell Line
HLA-C Antigens
Histocompatibility Antigens Class I
Humans
Mice
Molecular Dynamics Simulation
Oligopeptides
Peptides
Protein Binding
Protein Conformation
Respiratory Syncytial Virus Infections
Respiratory Syncytial Virus, Human
Descripción
Sumario:Short viral antigens bound to human major histocompatibility complex (HLA) class I molecules are presented on infected cells. Vaccine development frequently relies on synthetic peptides to identify optimal HLA class I ligands. However, when natural peptides are analyzed, more complex mixtures are found. By immunoproteomics analysis, we identify in this study a physiologically processed HLA ligand derived from the human respiratory syncytial virus matrix protein that is very different from what was expected from studies with synthetic peptides. This natural HLA-Cw4 class I ligand uses alternative interactions to the anchor motifs previously described for its presenting HLA-Cw4 class I molecule. Finally, this octameric peptide shares its C-terminal core with the H-2D(b) nonamer ligand previously identified in the mouse model. These data have implications for the identification of antiviral cytotoxic T lymphocyte responses and for vaccine development.