NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.

A role for the NADPH oxidases NOX1 and NOX2 in liver fibrosis has been proposed, but the implication of NOX4 is poorly understood yet. The aim of this work was to study the functional role of NOX4 in different cell populations implicated in liver fibrosis: hepatic stellate cells (HSC), myofibroblats...

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Autores: Sancho, Patrícia, Mainez Villoro, Jessica, Crosas Molist, Eva, Roncero, Cesáreo, Fernández Rodriguez, Conrado M., Pinedo, Fernando, Huber, Heidemarie, Eferl, Robert, Mikulits, Wolfgang, Fabregat Romero, Isabel
Formato: artículo
Estado:Versión publicada
Fecha de publicación:2012
País:España
Recursos:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
Repositorio:Recercat. Dipósit de la Recerca de Catalunya
OAI Identifier:oai:recercat.cat:2445/36383
Acesso em linha:https://hdl.handle.net/2445/36383
Access Level:acceso abierto
Palavra-chave:Malalties del fetge
Cèl·lules hepàtiques
Fibroblasts
Liver diseases
Liver cells
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repository_id_str
spelling NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.Sancho, PatríciaMainez Villoro, JessicaCrosas Molist, EvaRoncero, CesáreoFernández Rodriguez, Conrado M.Pinedo, FernandoHuber, HeidemarieEferl, RobertMikulits, WolfgangFabregat Romero, IsabelMalalties del fetgeCèl·lules hepàtiquesFibroblastsLiver diseasesLiver cellsFibroblastsA role for the NADPH oxidases NOX1 and NOX2 in liver fibrosis has been proposed, but the implication of NOX4 is poorly understood yet. The aim of this work was to study the functional role of NOX4 in different cell populations implicated in liver fibrosis: hepatic stellate cells (HSC), myofibroblats (MFBs) and hepatocytes. Two different mice models that develop spontaneous fibrosis (Mdr2−/−/p19ARF−/−, Stat3Δhc/Mdr2−/−) and a model of experimental induced fibrosis (CCl4) were used. In addition, gene expression in biopsies from chronic hepatitis C virus (HCV) patients or non-fibrotic liver samples was analyzed. Results have indicated that NOX4 expression was increased in the livers of all animal models, concomitantly with fibrosis development and TGF-β pathway activation. In vitro TGF-β-treated HSC increased NOX4 expression correlating with transdifferentiation to MFBs. Knockdown experiments revealed that NOX4 downstream TGF-β is necessary for HSC activation as well as for the maintenance of the MFB phenotype. NOX4 was not necessary for TGF-β-induced epithelial-mesenchymal transition (EMT), but was required for TGF-β-induced apoptosis in hepatocytes. Finally, NOX4 expression was elevated in patients with hepatitis C virus (HCV)-derived fibrosis, increasing along the fibrosis degree. In summary, fibrosis progression both in vitro and in vivo (animal models and patients) is accompanied by increased NOX4 expression, which mediates acquisition and maintenance of the MFB phenotype, as well as TGF-β-induced death of hepatocytes.Public Library of Science (PLoS)2013201320122013info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersion14 p.application/pdfapplication/pdfhttps://hdl.handle.net/2445/36383Articles publicats en revistes (Ciències Fisiològiques)reponame:Recercat. Dipósit de la Recerca de Catalunyainstname:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)InglésReproducció del document publicat a: http://dx.doi.org/10.1371/journal.pone.0045285PLoS One, 2012, vol. 7, num. 9, p. 1-14http://dx.doi.org/10.1371/journal.pone.0045285info:eu-repo/grantAgreement/EC/FP7/202047cc-by (c) Sancho, Patrícia et al., 2012http://creativecommons.org/licenses/by/3.0/esinfo:eu-repo/semantics/openAccessoai:recercat.cat:2445/363832026-05-29T05:05:01Z
dc.title.none.fl_str_mv NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.
title NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.
spellingShingle NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.
Sancho, Patrícia
Malalties del fetge
Cèl·lules hepàtiques
Fibroblasts
Liver diseases
Liver cells
Fibroblasts
title_short NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.
title_full NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.
title_fullStr NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.
title_full_unstemmed NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.
title_sort NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.
dc.creator.none.fl_str_mv Sancho, Patrícia
Mainez Villoro, Jessica
Crosas Molist, Eva
Roncero, Cesáreo
Fernández Rodriguez, Conrado M.
Pinedo, Fernando
Huber, Heidemarie
Eferl, Robert
Mikulits, Wolfgang
Fabregat Romero, Isabel
author Sancho, Patrícia
author_facet Sancho, Patrícia
Mainez Villoro, Jessica
Crosas Molist, Eva
Roncero, Cesáreo
Fernández Rodriguez, Conrado M.
Pinedo, Fernando
Huber, Heidemarie
Eferl, Robert
Mikulits, Wolfgang
Fabregat Romero, Isabel
author_role author
author2 Mainez Villoro, Jessica
Crosas Molist, Eva
Roncero, Cesáreo
Fernández Rodriguez, Conrado M.
Pinedo, Fernando
Huber, Heidemarie
Eferl, Robert
Mikulits, Wolfgang
Fabregat Romero, Isabel
author2_role author
author
author
author
author
author
author
author
author
dc.subject.none.fl_str_mv Malalties del fetge
Cèl·lules hepàtiques
Fibroblasts
Liver diseases
Liver cells
Fibroblasts
topic Malalties del fetge
Cèl·lules hepàtiques
Fibroblasts
Liver diseases
Liver cells
Fibroblasts
description A role for the NADPH oxidases NOX1 and NOX2 in liver fibrosis has been proposed, but the implication of NOX4 is poorly understood yet. The aim of this work was to study the functional role of NOX4 in different cell populations implicated in liver fibrosis: hepatic stellate cells (HSC), myofibroblats (MFBs) and hepatocytes. Two different mice models that develop spontaneous fibrosis (Mdr2−/−/p19ARF−/−, Stat3Δhc/Mdr2−/−) and a model of experimental induced fibrosis (CCl4) were used. In addition, gene expression in biopsies from chronic hepatitis C virus (HCV) patients or non-fibrotic liver samples was analyzed. Results have indicated that NOX4 expression was increased in the livers of all animal models, concomitantly with fibrosis development and TGF-β pathway activation. In vitro TGF-β-treated HSC increased NOX4 expression correlating with transdifferentiation to MFBs. Knockdown experiments revealed that NOX4 downstream TGF-β is necessary for HSC activation as well as for the maintenance of the MFB phenotype. NOX4 was not necessary for TGF-β-induced epithelial-mesenchymal transition (EMT), but was required for TGF-β-induced apoptosis in hepatocytes. Finally, NOX4 expression was elevated in patients with hepatitis C virus (HCV)-derived fibrosis, increasing along the fibrosis degree. In summary, fibrosis progression both in vitro and in vivo (animal models and patients) is accompanied by increased NOX4 expression, which mediates acquisition and maintenance of the MFB phenotype, as well as TGF-β-induced death of hepatocytes.
publishDate 2012
dc.date.none.fl_str_mv 2012
2013
2013
2013
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://hdl.handle.net/2445/36383
url https://hdl.handle.net/2445/36383
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv Reproducció del document publicat a: http://dx.doi.org/10.1371/journal.pone.0045285
PLoS One, 2012, vol. 7, num. 9, p. 1-14
http://dx.doi.org/10.1371/journal.pone.0045285
info:eu-repo/grantAgreement/EC/FP7/202047
dc.rights.none.fl_str_mv cc-by (c) Sancho, Patrícia et al., 2012
http://creativecommons.org/licenses/by/3.0/es
info:eu-repo/semantics/openAccess
rights_invalid_str_mv cc-by (c) Sancho, Patrícia et al., 2012
http://creativecommons.org/licenses/by/3.0/es
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv 14 p.
application/pdf
application/pdf
dc.publisher.none.fl_str_mv Public Library of Science (PLoS)
publisher.none.fl_str_mv Public Library of Science (PLoS)
dc.source.none.fl_str_mv Articles publicats en revistes (Ciències Fisiològiques)
reponame:Recercat. Dipósit de la Recerca de Catalunya
instname:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
instname_str Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
reponame_str Recercat. Dipósit de la Recerca de Catalunya
collection Recercat. Dipósit de la Recerca de Catalunya
repository.name.fl_str_mv
repository.mail.fl_str_mv
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