Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families

Bipolar disorder (BD) is a highly heritable neuropsychiatric disease characterized by recurrent episodes of depression and mania. Research suggests that the cumulative impact of common alleles explains 25-38% of phenotypic variance, and that rare variants may contribute to BD susceptibility. To iden...

ver descrição completa

Detalhes bibliográficos
Autores: Forstner, Andreas J., Fischer, Sascha B., Schenk, Lorena M., Strohmaier, Jana, Maaser-Hecker, Anna, Reinbold, Céline S., Sivalingam, Sugirthan, Hecker, Julian, Streit, Fabian, Degenhardt, Franziska, Witt, Stephanie H., Schumacher, Johannes, Thiele, Holger, Nürnberg, Peter, Guzman-Parra, José, Orozco Diaz, Guillermo, Auburger, Georg, Albus, Margot, Borrmann-Hassenbach, Margitta, González, Maria José, Gil Flores, Susana, Cabaleiro Fabeiro, Francisco J., del Río Noriega, Francisco, Perez Perez, Fermin, Haro González, Jesus, Rivas, Fabio, Mayoral, Fermin, Bauer, Michael, Pfennig, Andrea, Reif, Andreas, Herms, Stefan, Hoffmann, Per, Pirooznia, Mehdi, Goes, Fernando S., Rietschel, Marcella, Nöthen, Markus M., Cichon, Sven
Formato: artículo
Fecha de publicación:2020
País:España
Recursos:Instituto de Salud Carlos III (ISCIII)
Repositorio:Repisalud
Idioma:inglés
OAI Identifier:oai:repisalud.isciii.es:20.500.12105/17998
Acesso em linha:http://hdl.handle.net/20.500.12105/17998
Access Level:acceso abierto
Palavra-chave:Whole exome sequencing
Bipolar disorder
Neuropsychiatry
Genetic predisposition to disease
Secuenciación del exoma completo
Trastorno bipolar
Neuropsiquiatría
Predisposición genética a la enfermedad
Exome
Genetic Predisposition to Disease
Humans
Schizophrenia
Bipolar Disorder
Autistic Disorder
Spain
Germany
Penetrance
Brain
id ES_01fadcbd61e5d6dbffcca11f7b147ef0
oai_identifier_str oai:repisalud.isciii.es:20.500.12105/17998
network_acronym_str ES
network_name_str España
repository_id_str
dc.title.none.fl_str_mv Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families
title Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families
spellingShingle Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families
Forstner, Andreas J.
Whole exome sequencing
Bipolar disorder
Neuropsychiatry
Genetic predisposition to disease
Secuenciación del exoma completo
Trastorno bipolar
Neuropsiquiatría
Predisposición genética a la enfermedad
Exome
Genetic Predisposition to Disease
Humans
Schizophrenia
Bipolar Disorder
Autistic Disorder
Spain
Germany
Penetrance
Brain
title_short Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families
title_full Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families
title_fullStr Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families
title_full_unstemmed Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families
title_sort Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder families
dc.creator.none.fl_str_mv Forstner, Andreas J.
Fischer, Sascha B.
Schenk, Lorena M.
Strohmaier, Jana
Maaser-Hecker, Anna
Reinbold, Céline S.
Sivalingam, Sugirthan
Hecker, Julian
Streit, Fabian
Degenhardt, Franziska
Witt, Stephanie H.
Schumacher, Johannes
Thiele, Holger
Nürnberg, Peter
Guzman-Parra, José
Orozco Diaz, Guillermo
Auburger, Georg
Albus, Margot
Borrmann-Hassenbach, Margitta
González, Maria José
Gil Flores, Susana
Cabaleiro Fabeiro, Francisco J.
del Río Noriega, Francisco
Perez Perez, Fermin
Haro González, Jesus
Rivas, Fabio
Mayoral, Fermin
Bauer, Michael
Pfennig, Andrea
Reif, Andreas
Herms, Stefan
Hoffmann, Per
Pirooznia, Mehdi
Goes, Fernando S.
Rietschel, Marcella
Nöthen, Markus M.
Cichon, Sven
author Forstner, Andreas J.
author_facet Forstner, Andreas J.
Fischer, Sascha B.
Schenk, Lorena M.
Strohmaier, Jana
Maaser-Hecker, Anna
Reinbold, Céline S.
Sivalingam, Sugirthan
Hecker, Julian
Streit, Fabian
Degenhardt, Franziska
Witt, Stephanie H.
Schumacher, Johannes
Thiele, Holger
Nürnberg, Peter
Guzman-Parra, José
Orozco Diaz, Guillermo
Auburger, Georg
Albus, Margot
Borrmann-Hassenbach, Margitta
González, Maria José
Gil Flores, Susana
Cabaleiro Fabeiro, Francisco J.
del Río Noriega, Francisco
Perez Perez, Fermin
Haro González, Jesus
Rivas, Fabio
Mayoral, Fermin
Bauer, Michael
Pfennig, Andrea
Reif, Andreas
Herms, Stefan
Hoffmann, Per
Pirooznia, Mehdi
Goes, Fernando S.
Rietschel, Marcella
Nöthen, Markus M.
Cichon, Sven
author_role author
author2 Fischer, Sascha B.
Schenk, Lorena M.
Strohmaier, Jana
Maaser-Hecker, Anna
Reinbold, Céline S.
Sivalingam, Sugirthan
Hecker, Julian
Streit, Fabian
Degenhardt, Franziska
Witt, Stephanie H.
Schumacher, Johannes
Thiele, Holger
Nürnberg, Peter
Guzman-Parra, José
Orozco Diaz, Guillermo
Auburger, Georg
Albus, Margot
Borrmann-Hassenbach, Margitta
González, Maria José
Gil Flores, Susana
Cabaleiro Fabeiro, Francisco J.
del Río Noriega, Francisco
Perez Perez, Fermin
Haro González, Jesus
Rivas, Fabio
Mayoral, Fermin
Bauer, Michael
Pfennig, Andrea
Reif, Andreas
Herms, Stefan
Hoffmann, Per
Pirooznia, Mehdi
Goes, Fernando S.
Rietschel, Marcella
Nöthen, Markus M.
Cichon, Sven
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv [Forstner,AJ; Schumacher,J] Centre for Human Genetics, University of Marburg, Marburg, Germany. [Forstner,AJ; Schenk,LM; Maaser-Hecker,A; Sivalingam,S; Degenhardt,F; Schumacher,J; Herms,S; Hoffmann,P; Nöthen,MM; Cichon,S] Institute of Human Genetics, University of Bonn, School of Medicine & University Hospital Bonn, Bonn, Germany. [Forstner,AJ; Fischer,SB; Reinbold,CS; Herms,S; Hoffmann,P; Cichon,S] Department of Biomedicine, University of Basel, Basel, Switzerland. [Forstner,AJ] Department of Psychiatry (UPK), University of Basel, Basel, Switzerland. [Fischer,SB; Reinbold,CS; Herms,S; Hoffmann,P; Cichon,S] Institute of Medical Genetics and Pathology, University Hospital Basel, Basel, Switzerland. [Strohmaier,J; Streit,F; Witt,SH; Rietschel,M] Department of Genetic Epidemiology in Psychiatry, Central Institute of Mental Health, Medical Faculty Mannheim, University of Heidelberg, Mannheim, Germany. [Strohmaier,J] SRH University Heidelberg, Academy for Psychotherapy, Heidelberg, Germany. [Reinbold,CS] Center for Lifespan Changes in Brain and Cognition (LCBC), Department of Psychology, University of Oslo, Oslo, Norway. [Hecker,J] Department of Biostatistics, Harvard T.H. Chan School of Public Health, Boston, MA, USA. [Thiele,H; Nürnberg,P] Cologne Center for Genomics, University of Cologne, Cologne, Germany. [Guzman-Parra,J; González,MJ] Department of Mental Health, University Regional Hospital of Málaga, Institute of Biomedicine of Málaga (IBIMA), Málaga, Spain. [Orozco Diaz,G] Unidad de Gestión Clínica del Dispositivo de Cuidados Críticos y Urgencias del Distrito Sanitario Málaga - Coin- Gudalhorce, Málaga, Spain. [Auburger,G] Experimental Neurology, Department of Neurology, Goethe University Hospital, Frankfurt am Main, Germany. [Albus,M; Borrmann-Hassenbach,M]Isar Amper Klinikum München Ost, kbo, Haar, Germany. [Gil Flores,S] Department of Mental Health, University Hospital of Reina Sofia, Cordoba, Spain. [Cabaleiro Fabeiro,FJ] Department of Mental Health, Hospital of Jaén, Jaén, Spain. [del Río Noriega,F] Department of Mental Health, Hospital of Jerez de la Frontera, Jerez de la Frontera, Spain. [Perez Perez,F] Department of Mental Health, Hospital of Puerto Real, Cádiz, Spain. [Haro González,J] Department of Mental Health, Hospital Punta de Europa, Algeciras, Spain. [Rivas,F; Mayoral,F] Department of Psychiatry, Carlos Haya Regional University Hospital, Malaga, Spain. [Bauer,M; Pfennig,A] Department of Psychiatry and Psychotherapy, Medical Faculty, University Hospital Carl Gustav Carus, Technische Universität Dresden, Dresden, Germany. [Reif,A] Department of Psychiatry, Psychosomatic Medicine and Psychotherapy, University Hospital Frankfurt am Main, Frankfurt am Main, Germany. [Hoffmann,P; Cichon,S] Institute of Neuroscience and Medicine (INM-1), Research Center Jülich, Jülich, Germany. [Pirooznia,M; Goes,FS] Department of Psychiatry and Behavioral Sciences, Johns Hopkins University School of Medicine, Baltimore, MD, USA

dc.subject.none.fl_str_mv Whole exome sequencing
Bipolar disorder
Neuropsychiatry
Genetic predisposition to disease
Secuenciación del exoma completo
Trastorno bipolar
Neuropsiquiatría
Predisposición genética a la enfermedad
Exome
Genetic Predisposition to Disease
Humans
Schizophrenia
Bipolar Disorder
Autistic Disorder
Spain
Germany
Penetrance
Brain
topic Whole exome sequencing
Bipolar disorder
Neuropsychiatry
Genetic predisposition to disease
Secuenciación del exoma completo
Trastorno bipolar
Neuropsiquiatría
Predisposición genética a la enfermedad
Exome
Genetic Predisposition to Disease
Humans
Schizophrenia
Bipolar Disorder
Autistic Disorder
Spain
Germany
Penetrance
Brain
description Bipolar disorder (BD) is a highly heritable neuropsychiatric disease characterized by recurrent episodes of depression and mania. Research suggests that the cumulative impact of common alleles explains 25-38% of phenotypic variance, and that rare variants may contribute to BD susceptibility. To identify rare, high-penetrance susceptibility variants for BD, whole-exome sequencing (WES) was performed in three affected individuals from each of 27 multiply affected families from Spain and Germany. WES identified 378 rare, non-synonymous, and potentially functional variants. These spanned 368 genes, and were carried by all three affected members in at least one family. Eight of the 368 genes harbored rare variants that were implicated in at least two independent families. In an extended segregation analysis involving additional family members, five of these eight genes harbored variants showing full or nearly full cosegregation with BD. These included the brain-expressed genes RGS12 and NCKAP5, which were considered the most promising BD candidates on the basis of independent evidence. Gene enrichment analysis for all 368 genes revealed significant enrichment for four pathways, including genes reported in de novo studies of autism (padj < 0.006) and schizophrenia (padj = 0.015). These results suggest a possible genetic overlap with BD for autism and schizophrenia at the rare-sequence-variant level. The present study implicates novel candidate genes for BD development, and may contribute to an improved understanding of the biological basis of this common and often devastating disease.
publishDate 2020
dc.date.none.fl_str_mv 2020
2020-02-04
2020
2020-02-04
2024
2024-02-12
dc.type.none.fl_str_mv research article
http://purl.org/coar/resource_type/c_2df8fbb1
VoR
http://purl.org/coar/version/c_970fb48d4fbd8a85
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv http://hdl.handle.net/20.500.12105/17998
url http://hdl.handle.net/20.500.12105/17998
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution 4.0 International
http://creativecommons.org/licenses/by/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution 4.0 International
http://creativecommons.org/licenses/by/4.0/
eu_rights_str_mv openAccess
dc.publisher.none.fl_str_mv Springer
publisher.none.fl_str_mv Springer
dc.source.none.fl_str_mv reponame:Repisalud
instname:Instituto de Salud Carlos III (ISCIII)
instname_str Instituto de Salud Carlos III (ISCIII)
reponame_str Repisalud
collection Repisalud
repository.name.fl_str_mv
repository.mail.fl_str_mv
_version_ 1869402622633443328
spelling Whole-exome sequencing of 81 individuals from 27 multiply affected bipolar disorder familiesForstner, Andreas J.Fischer, Sascha B.Schenk, Lorena M.Strohmaier, JanaMaaser-Hecker, AnnaReinbold, Céline S.Sivalingam, SugirthanHecker, JulianStreit, FabianDegenhardt, FranziskaWitt, Stephanie H.Schumacher, JohannesThiele, HolgerNürnberg, PeterGuzman-Parra, JoséOrozco Diaz, GuillermoAuburger, GeorgAlbus, MargotBorrmann-Hassenbach, MargittaGonzález, Maria JoséGil Flores, SusanaCabaleiro Fabeiro, Francisco J.del Río Noriega, FranciscoPerez Perez, FerminHaro González, JesusRivas, FabioMayoral, FerminBauer, MichaelPfennig, AndreaReif, AndreasHerms, StefanHoffmann, PerPirooznia, MehdiGoes, Fernando S.Rietschel, MarcellaNöthen, Markus M.Cichon, SvenWhole exome sequencingBipolar disorderNeuropsychiatryGenetic predisposition to diseaseSecuenciación del exoma completoTrastorno bipolarNeuropsiquiatríaPredisposición genética a la enfermedadExomeGenetic Predisposition to DiseaseHumansSchizophreniaBipolar DisorderAutistic DisorderSpainGermanyPenetranceBrainBipolar disorder (BD) is a highly heritable neuropsychiatric disease characterized by recurrent episodes of depression and mania. Research suggests that the cumulative impact of common alleles explains 25-38% of phenotypic variance, and that rare variants may contribute to BD susceptibility. To identify rare, high-penetrance susceptibility variants for BD, whole-exome sequencing (WES) was performed in three affected individuals from each of 27 multiply affected families from Spain and Germany. WES identified 378 rare, non-synonymous, and potentially functional variants. These spanned 368 genes, and were carried by all three affected members in at least one family. Eight of the 368 genes harbored rare variants that were implicated in at least two independent families. In an extended segregation analysis involving additional family members, five of these eight genes harbored variants showing full or nearly full cosegregation with BD. These included the brain-expressed genes RGS12 and NCKAP5, which were considered the most promising BD candidates on the basis of independent evidence. Gene enrichment analysis for all 368 genes revealed significant enrichment for four pathways, including genes reported in de novo studies of autism (padj < 0.006) and schizophrenia (padj = 0.015). These results suggest a possible genetic overlap with BD for autism and schizophrenia at the rare-sequence-variant level. The present study implicates novel candidate genes for BD development, and may contribute to an improved understanding of the biological basis of this common and often devastating disease.Springer[Forstner,AJ; Schumacher,J] Centre for Human Genetics, University of Marburg, Marburg, Germany. [Forstner,AJ; Schenk,LM; Maaser-Hecker,A; Sivalingam,S; Degenhardt,F; Schumacher,J; Herms,S; Hoffmann,P; Nöthen,MM; Cichon,S] Institute of Human Genetics, University of Bonn, School of Medicine & University Hospital Bonn, Bonn, Germany. [Forstner,AJ; Fischer,SB; Reinbold,CS; Herms,S; Hoffmann,P; Cichon,S] Department of Biomedicine, University of Basel, Basel, Switzerland. [Forstner,AJ] Department of Psychiatry (UPK), University of Basel, Basel, Switzerland. [Fischer,SB; Reinbold,CS; Herms,S; Hoffmann,P; Cichon,S] Institute of Medical Genetics and Pathology, University Hospital Basel, Basel, Switzerland. [Strohmaier,J; Streit,F; Witt,SH; Rietschel,M] Department of Genetic Epidemiology in Psychiatry, Central Institute of Mental Health, Medical Faculty Mannheim, University of Heidelberg, Mannheim, Germany. [Strohmaier,J] SRH University Heidelberg, Academy for Psychotherapy, Heidelberg, Germany. [Reinbold,CS] Center for Lifespan Changes in Brain and Cognition (LCBC), Department of Psychology, University of Oslo, Oslo, Norway. [Hecker,J] Department of Biostatistics, Harvard T.H. Chan School of Public Health, Boston, MA, USA. [Thiele,H; Nürnberg,P] Cologne Center for Genomics, University of Cologne, Cologne, Germany. [Guzman-Parra,J; González,MJ] Department of Mental Health, University Regional Hospital of Málaga, Institute of Biomedicine of Málaga (IBIMA), Málaga, Spain. [Orozco Diaz,G] Unidad de Gestión Clínica del Dispositivo de Cuidados Críticos y Urgencias del Distrito Sanitario Málaga - Coin- Gudalhorce, Málaga, Spain. [Auburger,G] Experimental Neurology, Department of Neurology, Goethe University Hospital, Frankfurt am Main, Germany. [Albus,M; Borrmann-Hassenbach,M]Isar Amper Klinikum München Ost, kbo, Haar, Germany. [Gil Flores,S] Department of Mental Health, University Hospital of Reina Sofia, Cordoba, Spain. [Cabaleiro Fabeiro,FJ] Department of Mental Health, Hospital of Jaén, Jaén, Spain. [del Río Noriega,F] Department of Mental Health, Hospital of Jerez de la Frontera, Jerez de la Frontera, Spain. [Perez Perez,F] Department of Mental Health, Hospital of Puerto Real, Cádiz, Spain. [Haro González,J] Department of Mental Health, Hospital Punta de Europa, Algeciras, Spain. [Rivas,F; Mayoral,F] Department of Psychiatry, Carlos Haya Regional University Hospital, Malaga, Spain. [Bauer,M; Pfennig,A] Department of Psychiatry and Psychotherapy, Medical Faculty, University Hospital Carl Gustav Carus, Technische Universität Dresden, Dresden, Germany. [Reif,A] Department of Psychiatry, Psychosomatic Medicine and Psychotherapy, University Hospital Frankfurt am Main, Frankfurt am Main, Germany. [Hoffmann,P; Cichon,S] Institute of Neuroscience and Medicine (INM-1), Research Center Jülich, Jülich, Germany. [Pirooznia,M; Goes,FS] Department of Psychiatry and Behavioral Sciences, Johns Hopkins University School of Medicine, Baltimore, MD, USA20242024-02-1220202020-02-0420202020-02-04research articlehttp://purl.org/coar/resource_type/c_2df8fbb1VoRhttp://purl.org/coar/version/c_970fb48d4fbd8a85info:eu-repo/semantics/articlehttp://hdl.handle.net/20.500.12105/17998reponame:Repisaludinstname:Instituto de Salud Carlos III (ISCIII)Inglésengopen accesshttp://purl.org/coar/access_right/c_abf2Attribution 4.0 Internationalhttp://creativecommons.org/licenses/by/4.0/info:eu-repo/semantics/openAccessoai:repisalud.isciii.es:20.500.12105/179982026-06-12T12:43:37Z
score 15,812429