Resistant Hypertension: A Clinical Case
Introduction: Secondary hypertension corresponds to 15 % of the causes of arterial hypertension, and among them, primary hyperaldosteronism presents a variable incidence of about 3% in hypertensive patients. It has a slightly higher prevalence in women, between 30 and 60 years, and is usually unilat...
| Autores: | , , , |
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| Tipo de recurso: | artículo |
| Estado: | Versión publicada |
| Fecha de publicación: | 2018 |
| País: | Colombia |
| Institución: | Universidad del Rosario |
| Repositorio: | Repositorio EdocUR - U. Rosario |
| Idioma: | español inglés |
| OAI Identifier: | oai:repository.urosario.edu.co:10336/29462 |
| Acceso en línea: | https://doi.org/10.12804/revistas.urosario.edu.co/revsalud/a.7272 https://repository.urosario.edu.co/handle/10336/29462 |
| Access Level: | acceso abierto |
| Palabra clave: | Adenoma de Conn Adenoma adrenal Hipertensión arterial Hipertensión secundaria Aldosteronismo primario Hipertensión resistente Cardiología Conn's adenoma Adrenal adenoma Arterial hypertension Secondary hypertension Primary aldosteronism Resistant hypertension Cardiology Hipertensão arterial Hipertensão secundária Aldos-teronismo primário Hipertensão resistente Cardiologia |
| Sumario: | Introduction: Secondary hypertension corresponds to 15 % of the causes of arterial hypertension, and among them, primary hyperaldosteronism presents a variable incidence of about 3% in hypertensive patients. It has a slightly higher prevalence in women, between 30 and 60 years, and is usually unilateral. Case presentation: The authors describe the clinical case of a patient, followed by severe hypertension medicated with four antihypertensive drugs for tension stabilization, maintaining systolic arterial tensions superior to 170 mmHg. In the aetiological study of hypertension, analytical alterations suggested hyperaldosteronism and a nodular lesion was detected in the left adrenal gland. The patient was submitted to surgery and excision of the lesion was done with histological confirmation of the diagnosis of corticomedullary adenoma of the adrenal gland. The patient presented improvement of the tension profile, with the need to suspend two of the four antihypertensive drugs and to reduce the dose of the remaining ones. Conclusion: A tumor of the adrenal cortex producing aldosterone is the main cause of primary hyperaldosteronism and should always be excluded when the presence of di cult to control, severe hypertension is detected, since the standard treatment is surgical, leading to a stabilization of the tension pattern after a few months. |
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