Profiling of gene expression regulated by 17 beta-estradiol and tamoxifen in estrogen receptor-positive and estrogen receptor-negative human breast cancer cell lines

One area of great importance in breast cancer (BC) research is the study of gene expression regulated by both estrogenic and antiestrogenic agents. Although many studies have been performed in this area, most of them have only addressed the effects of 17 beta-estradiol (E2) and tamoxifen (TAM) on MC...

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Detalhes bibliográficos
Autores: Rangel, Nelson, Villegas Gálvez, Victoria Eugenia, Rondon-Lagos, Milena
Formato: artículo
Estado:Versión publicada
Fecha de publicación:2017
País:Colombia
Recursos:Universidad del Rosario
Repositorio:Repositorio EdocUR - U. Rosario
Idioma:inglés
OAI Identifier:oai:repository.urosario.edu.co:10336/24963
Acesso em linha:https://doi.org/10.2147/BCTT.S146247
https://repository.urosario.edu.co/handle/10336/24963
Access Level:acceso abierto
Palavra-chave:cáncer de mama
líneas celulares
17 beta-estradiol
tamoxifeno
ER alfa +
ER alfa (-)
qPCR
Análisis integrador
Cáncer colonrectal
Charla cruzada
Alfa
Beta
Mecanismos
Crecimiento
Proliferación
Patrones
Proteína
breast cancer
cell lines
tamoxifen
ER alpha+
ER alpha(-)
Integrative analysis
Colorectal-cancer
Cross-talk
Alpha
Mechanisms
Growth
Proliferation
Patterns
Protein
Descrição
Resumo:One area of great importance in breast cancer (BC) research is the study of gene expression regulated by both estrogenic and antiestrogenic agents. Although many studies have been performed in this area, most of them have only addressed the effects of 17 beta-estradiol (E2) and tamoxifen (TAM) on MCF7 cells. This study aimed to determine the effect of low doses of E2 and TAM on the expression levels of 84 key genes, which are commonly involved in breast carcinogenesis, in four BC cell lines differentially expressing estrogen receptor (ER) alpha and HER2 (MCF7, T47D, BT474, and SKBR3). The results allowed us to determine the expression patterns modulated by E2 and TAM in ER alpha+ and ER alpha(-)cell lines, as well as to identify differences in expression patterns. Although the MCF7 cell line is the most frequently used model to determine gene expression profiles in response to E2 and TAM, the changes in gene expression patterns identified in ER alpha+ and ER alpha(-)cell lines could reflect distinctive properties of these cells. Our results could provide important markers to be validated in BC patient samples, and subsequently used for predicting the outcome in ER alpha+ and ER alpha(-)tumors after TAM or hormonal therapy. Considering that BC is a molecularly heterogeneous disease, it is important to understand how well, and which cell lines, best model that diversity.