Angiotensin ii contributes to renal fibrosis independently of notch pathway activation

14

Detalhes bibliográficos
Autores: Lavoz, Carolina, Rodrigues-Diez, Raquel, Benito-Martin, Alberto, Rayego-Mateos, Sandra, Rodrigues-Diez, Raul R., Alique, Matilde, Ortiz, Alberto, Mezzano, Sergio, Egido, Jesus, Ruiz-Ortega, Marta
Tipo de documento: artigo
Estado:Versão publicada
Data de publicação:2012
País:Chile
Idioma:inglês
OAI Identifier:oai:repositorio.anid.cl:10533/138347
Acesso em linha:https://hdl.handle.net/10533/138347
Access Level:Acceso aberto
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spelling Ruiz-Ortega, MartaEgido, JesusMezzano, SergioOrtiz, AlbertoAlique, MatildeRodrigues-Diez, Raul R.Rayego-Mateos, SandraBenito-Martin, AlbertoRodrigues-Diez, RaquelLavoz, Carolina201210.1371/journal.pone.0040490https://hdl.handle.net/10533/138347http://purl.org/coar/access_right/c_abf2Angiotensin ii contributes to renal fibrosis independently of notch pathway activationusaSAN FRANCISCOLavoz, CarolinaRodrigues-Diez, RaquelBenito-Martin, AlbertoRayego-Mateos, SandraRodrigues-Diez, Raul R.Alique, MatildeOrtiz, AlbertoMezzano, SergioEgido, JesusRuiz-Ortega, Marta2016-12-05T14:55:17Z2022-07-06T17:21:06Z2016-12-05T14:55:17Z2022-07-06T17:21:06Z201214FONDECYTmruizo@fjd.es7FONDECYT711204801120480virtual::38994-1WOS:0003063547000561932-6203https://hdl.handle.net/10533/138347engPUBLIC LIBRARY SCIENCEinstname: Conicytreponame: Repositorio Digital RI2.0instname: Conicytreponame: Repositorio Digital RI2.010.1371/journal.pone.0040490info:eu-repo/grantAgreement/Fondecyt/1120480info:eu-repo/semantics/dataset/hdl.handle.net/10533/93477https://doi.org/10.1371/journal.pone.0040490info:eu-repo/semantics/openAccessAngiotensin ii contributes to renal fibrosis independently of notch pathway activationPLOS ONEPLOS ONEArticuloinfo:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionArticulohttps://hdl.handle.net/10533/138347FONDECYThttp://purl.org/coar/resource_type/c_2df8fbb187da8d01-8a7e-43b6-a7c1-d8f8fdce6ec3virtual::38994-187da8d01-8a7e-43b6-a7c1-d8f8fdce6ec3virtual::38994-110533/138347oai:repositorio.anid.cl:10533/1383472023-07-24 16:19:54.056https://repositorio.anid.clRepositorio ANIDaletelier@anid.cl
dc.title.none.fl_str_mv Angiotensin ii contributes to renal fibrosis independently of notch pathway activation
dc.title.journal.none.fl_str_mv PLOS ONE
dc.title.journalabbreviation.none.fl_str_mv PLOS ONE
title Angiotensin ii contributes to renal fibrosis independently of notch pathway activation
spellingShingle Angiotensin ii contributes to renal fibrosis independently of notch pathway activation
Lavoz, Carolina
title_short Angiotensin ii contributes to renal fibrosis independently of notch pathway activation
title_full Angiotensin ii contributes to renal fibrosis independently of notch pathway activation
title_fullStr Angiotensin ii contributes to renal fibrosis independently of notch pathway activation
title_full_unstemmed Angiotensin ii contributes to renal fibrosis independently of notch pathway activation
title_sort Angiotensin ii contributes to renal fibrosis independently of notch pathway activation
dc.creator.none.fl_str_mv Lavoz, Carolina
Rodrigues-Diez, Raquel
Benito-Martin, Alberto
Rayego-Mateos, Sandra
Rodrigues-Diez, Raul R.
Alique, Matilde
Ortiz, Alberto
Mezzano, Sergio
Egido, Jesus
Ruiz-Ortega, Marta
author Lavoz, Carolina
author_facet Lavoz, Carolina
Rodrigues-Diez, Raquel
Benito-Martin, Alberto
Rayego-Mateos, Sandra
Rodrigues-Diez, Raul R.
Alique, Matilde
Ortiz, Alberto
Mezzano, Sergio
Egido, Jesus
Ruiz-Ortega, Marta
author_role author
author2 Rodrigues-Diez, Raquel
Benito-Martin, Alberto
Rayego-Mateos, Sandra
Rodrigues-Diez, Raul R.
Alique, Matilde
Ortiz, Alberto
Mezzano, Sergio
Egido, Jesus
Ruiz-Ortega, Marta
author2_role author
author
author
author
author
author
author
author
author
description 14
publishDate 2012
dc.date.issued.none.fl_str_mv 2012
dc.date.accessioned.none.fl_str_mv 2016-12-05T14:55:17Z
2022-07-06T17:21:06Z
dc.date.available.none.fl_str_mv 2016-12-05T14:55:17Z
2022-07-06T17:21:06Z
dc.type.none.fl_str_mv Articulo
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dc.relation.doi.none.fl_str_mv 10.1371/journal.pone.0040490
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