Modulation of ryanodine receptor channels from rat brain cortex in lipid bilayers.
In this work we show that caffeine, calcium, ATP, and cADPR activate RyR channels from rat brain cortex. Oxidation of SH residues increases the apparent affinity for agonists such as calcium (Marengo et al., 1998) and ATP (Bull et al., 2003). Furthermore,
| Autores: | , , , |
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| Tipo de recurso: | capítulo de libro |
| Estado: | Versión publicada |
| Fecha de publicación: | 2005 |
| País: | Chile |
| Idioma: | inglés |
| OAI Identifier: | oai:repositorio.anid.cl:10533/242305 |
| Acceso en línea: | https://hdl.handle.net/10533/242305 |
| Access Level: | acceso abierto |
| Sumario: | In this work we show that caffeine, calcium, ATP, and cADPR activate RyR channels from rat brain cortex. Oxidation of SH residues increases the apparent affinity for agonists such as calcium (Marengo et al., 1998) and ATP (Bull et al., 2003). Furthermore, |
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