Chromosome 17 abnormalities and mutation of the TP53 gene: Correlation between cytogenetics, flow cytometry and molecular analysis in three cases of chronic myeloid leukemia

Alterations involving the short arm of chromosome 17 (17p) during the progression of chronic myeloid leukemia (CML) have been described. This chromosomal region contains the tumor suppressor gene TP53 that may be an important factor in the evolution of this disease. In this study, we used flow cytom...

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Detalles Bibliográficos
Autores: Otero, Luize, Cavalcanti Júnior, Geraldo Barroso, Klumb, Claudete Esteves, Scheiner, Marcos Antonio Mauricio, Magluta, Eliane Pereira Simões, Fernandez, Teresa de Souza, Silva, Maria Luiza Macedo, Pires, Virgínia, Andrade, Gabriela Vasconcelos, Maia, Raquel Ciuvalschi, Tabak, Daniel
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2005
País:Brasil
Institución:Universidade Federal do Rio Grande do Norte (UFRN)
Repositorio:Repositório Institucional da UFRN
Idioma:portugués
OAI Identifier:oai:repositorio.ufrn.br:123456789/40854
Acceso en línea:https://repositorio.ufrn.br/handle/123456789/40854
Access Level:acceso abierto
Palabra clave:Chronic myeloid leukemia
Chromosome 17
TP53 gene
P53 protein
Descripción
Sumario:Alterations involving the short arm of chromosome 17 (17p) during the progression of chronic myeloid leukemia (CML) have been described. This chromosomal region contains the tumor suppressor gene TP53 that may be an important factor in the evolution of this disease. In this study, we used flow cytometry and western blotting to assess p53 protein expression and single stranded conformational polymorphism to examine TP53 gene alterations in three patients with CML who showed alterations in 17p. Only the case with del(17)(p11) had p53 expression positive by flow cytometry and an abnormal migration pattern by SSCP analysis. The importance of the correlation between the results obtained with these techniques, as well as the clinical course of the patients, are discussed.