Bibliographic survey of the mechanism of development of Type II Diabetes Mellitus after infection by SARS-COV-2

This study aims to conduct a systematic review of the literature to understand the mechanism of development of type II DM after SARS-COV-2 infection and to point out new updates on the subject. Currently, the world faces the pandemic of Coronavirus Disease 2019, a severe acute respiratory syndrome c...

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Detalles Bibliográficos
Autores: Carvalho, Felipe dos Santos, Machado, Eliza Caetano, Leitão, Agnaldo Saraiva, Ramos, Dandara Franco, Oliveira, Laís Túlio, Lima , Ana Julia Aidar, Mendes, Guilherme Stephano Moreira
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2022
País:Brasil
Institución:Universidade Federal de Itajubá (UNIFEI)
Repositorio:Research, Society and Development
Idioma:portugués
OAI Identifier:oai:ojs.pkp.sfu.ca:article/33999
Acceso en línea:https://rsdjournal.org/index.php/rsd/article/view/33999
Access Level:acceso abierto
Palabra clave:Diabetes mellitus tipo II
SARS-COV-2
Disfunción metabólica de la glucosa.
SARS-CoV-2
Disfunção metabólica glicose.
Diabetes mellitus type II
Metabolic glucose dysfunction.
Descripción
Sumario:This study aims to conduct a systematic review of the literature to understand the mechanism of development of type II DM after SARS-COV-2 infection and to point out new updates on the subject. Currently, the world faces the pandemic of Coronavirus Disease 2019, a severe acute respiratory syndrome caused by coronavirus SARS-COV-2, having as one of the most frequent pathological consequences diabetes mellitus type II (Type II DM). DM type II is a metabolic endocrine dysfunction characterized by excess circulating glucose in the blood due to defects in the secretion and/or action of insulin, thus hindering the use of glucose as an energy source for the homeostatic metabolic functions of the human body. To date, several epidemiological studies indicate that a high pathological relationship between Covid-19 and DM type II where both pathologies complement each other to generate simultaneous harmful damage. By bringing pancreatic beta cell dysfunction, SARS-COV-2 can induce transient acute hyperglycemia or lead to a genetically predisposed individual developing DM type II. The prevalence of DM type II in patients recovering from post-covid 19 infection is notorious, but new searches to better understand its etiology are crucial.