Quantitative and molecular analysis of noroviruses RNA in blood from children hospitalized for acute gastroenteritis in Belém, Brazil

Background: Noroviruses (NoVs) are a common cause of acute gastroenteritis (AGE) and until now, littleis known about its ability to spread outside the gut. Objectives: We aim to investigate the role of NoVs causing viremia in children hospitalized for AGE, aswell as to correlate the presence of NoVs...

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Detalhes bibliográficos
Autores: Fumian, Tulio Machado, Justino, Maria Cleonice Aguiar, Mascarenhas, Joana D'Arc Pereira, Reymão, Tammy K. A, Abreu, Erika, Soares, Luana, Linhares, Alexandre da Costa, Gabbay, Yvone Benchimol
Tipo de documento: artigo
Estado:Versão publicada
Data de publicação:2013
País:Brasil
Recursos:Instituto Evandro Chagas (IEC)
Repositório:Repositório Digital do Instituto Evandro Chagas (Patuá)
Idioma:inglês
OAI Identifier:oai:patua.iec.gov.br:iec/3275
Acesso em linha:https://patua.iec.gov.br/handle/iec/3275
Access Level:Acceso aberto
Palavra-chave:Norovirus
Viremia
Carga Viral
Descrição
Resumo:Background: Noroviruses (NoVs) are a common cause of acute gastroenteritis (AGE) and until now, littleis known about its ability to spread outside the gut. Objectives: We aim to investigate the role of NoVs causing viremia in children hospitalized for AGE, aswell as to correlate the presence of NoVs RNA in serum with clinical severity and stool viral load. Study design: Paired stool and serum samples were collected from 85 pediatric patients under 6 years hospitalized for AGE from March to September 2012 in Belém, Brazil. Enzyme-linked immunosorbent assay (EIA) and reverse transcription quantitative PCR (RT-qPCR) were used to detect and quantify NoVs, respectively. Phylogenetic analysis of the partial ORF2 region was used to genotype the strains detected. Results: NoVs were detected in 34.1 per cent (29/85) of stool samples. By qRT-PCR, we found a high rate of NoVsÆ RNA in serum samples (34.5 per cent) among NoVs-positive AGE cases, and was associated with a longer hospitalization (6.5 vs. 4.0 days; p = 0.006), as well as with a higher stool viral load (3.9 Î 1011vs. 1.1 Î 1011GC/g; p = 0.0472). NoVs strains were classified as GII.4 (90 per cent of genotyped strains) and GII.7(10 per cent). The same genotype was found in paired stool and serum samples. Conclusion: Detection and molecular characterization of NoVs GII in paired stool and serum samples suggest that the dissemination of NoVs to the blood stream is not uncommon, but the role of viruses spread outside the gut and the relationship with disease severity need to be further addressed.