Immunophenotypical and pathological changes in dogs experimentally infected with Ehrlichia canis

Canine monocytic ehrlichiosis (CME) is one of the most important tick-borne diseases worldwide, with multisystemic presentations. Immune dysregulation has been proposed as the primary mechanism involved in its pathogenesis and in tissue injury in dogs with CME. Experimental infection of German Sheph...

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Detalles Bibliográficos
Autores: Castro, Márcio Botelho de, Szabó, Matias Pablo Juan, Aquino, Lucia Padilha Cury Thomaz de, Dagnoni, Ana Silvia, Alessi, Antonio Carlos [UNESP], Costa, Mirela Tinucci [UNESP], Nakaghi, Andréa Cristina Higa, Santi, Mariele De [UNESP], Calchi, Ana Claúdia [UNESP], André, Marcos Rogério [UNESP], Machado, Rosangela Zacarias [UNESP]
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2022
País:Brasil
Institución:Universidade Estadual Paulista (UNESP)
Repositorio:Repositório Institucional da UNESP
Idioma:inglés
OAI Identifier:oai:repositorio.unesp.br:11449/240831
Acceso en línea:http://dx.doi.org/10.1590/S1984-29612022020
http://hdl.handle.net/11449/240831
Access Level:acceso abierto
Palabra clave:canine
immunity
infection
Monocytic ehrlichiosis
tick-borne disease
Descripción
Sumario:Canine monocytic ehrlichiosis (CME) is one of the most important tick-borne diseases worldwide, with multisystemic presentations. Immune dysregulation has been proposed as the primary mechanism involved in its pathogenesis and in tissue injury in dogs with CME. Experimental infection of German Shepherd dogs in the present study demonstrated that CME caused marked pathological changes in their lymph nodes and spleen, and also gave rise to mononuclear infiltration in organs and tissues. Immunophenotyping of cells in lymph nodes, spleen and injured tissues highlighted differences in lymphocyte subsets, local expression of immunoglobulin subclasses and MHCII molecules between infected and control dogs. These findings suggest that the immunophenotypic and immunopathological changes in dogs with acute experimental CME are related to Th1 bias and compartmentalized immune response.