Alpha-tocopheryl succinate and doxorubicin-loaded liposomes improve drug uptake and tumor accumulation in a murine breast tumor model

Liposomes composed of a rigid bilayer have high plasma stability; however, they can be challenged in efficacy due to complications in releasing the encapsulated drug as well as being internalized by the tumor cell. On the other hand, fusogenic liposomes may fuse with the plasmatic membrane and relea...

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Detalles Bibliográficos
Autores: Fernanda Boratto, Eduardo Lages, Cristina Loures, Adriano Sabino, Angelo Malachias, Danyelle Townsend, André Luís Branco de Barros, Lucas Antonio Miranda Ferreira, Elaine Amaral Leite
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2023
País:Brasil
Institución:Universidade Federal de Minas Gerais (UFMG)
Repositorio:Repositório Institucional da UFMG
Idioma:inglés
OAI Identifier:oai:repositorio.ufmg.br:1843/79648
Acceso en línea:https://doi.org/10.1016/j.biopha.2023.115034
http://hdl.handle.net/1843/79648
https://orcid.org/0000-0001-6704-4057
https://orcid.org/0000-0002-2912-5312
https://orcid.org/0000-0001-8562-8689
https://orcid.org/0000-0002-8703-4283
https://orcid.org/0000-0001-7979-1156
https://orcid.org/0000-0003-4782-5416
Access Level:acceso abierto
Palabra clave:Doxorubicin
Alpha-tocopheryl succinate
PH-sensitive liposomes
Tumor accumulation
Breast cancer
Câncer
Tumores
Biologia celular e molecular
Descripción
Sumario:Liposomes composed of a rigid bilayer have high plasma stability; however, they can be challenged in efficacy due to complications in releasing the encapsulated drug as well as being internalized by the tumor cell. On the other hand, fusogenic liposomes may fuse with the plasmatic membrane and release encapsulated material directly into the cytoplasm. In a previous study, fusogenic liposomes composed of alpha-tocopheryl succinate (TS) and doxorubicin (DOX) were developed (pHSL-TS-DOX). These stabilized tumor growth and reduced toxicity compared to a commercial formulation. In the present study, we investigated whether cellular uptake or DOX accumulation in the tumor could justify the better performance of the pHSL-TS-DOX formulation. Release, deformability, and DOX plasmatic concentration studies were also carried out. pHSL-TS-DOX showed an adequate release profile and demonstrated characteristics of a deformable formulation. Data from apoptosis, cell cycle, and nuclear morphology studies have shown that the induction of cell death caused by pHSL-TS-DOX occurred more quickly. Higher DOX cellular uptake and tumor accumulation were observed when pHSL-TSDOX was administered, demonstrating better drug delivery capacity. Therefore, better DOX uptake as well as tumor accumulation explain the great antitumor activity previously demonstrated for this formulation.