TNFA-308G>A and IL10-1082A>G polymorphisms seem to be predictive biomarkers of chronic HCV infection

Background: Genetic changes may induce dysregulated cytokine production and afect the progression of the chronic disease caused by the hepacivirus C (HCV) because the balance of pro- and anti-infammatory cytokines determines the outcome of infection. This study evaluated the TNFA -308G>A and IL10...

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Detalles Bibliográficos
Autores: Santiago, Angélica Menezes, Amoras, Ednelza da Silva Graça, Queiroz, Maria Alice Freitas, Conde, Simone Regina Souza da Silva, Cayres-Vallinoto, Izaura Maria Vieira, Ishak, Ricardo, Vallinoto, Antonio Carlos Rosário
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2021
País:Brasil
Institución:Instituto Evandro Chagas (IEC)
Repositorio:Repositório Digital do Instituto Evandro Chagas (Patuá)
Idioma:inglés
OAI Identifier:oai:patua.iec.gov.br:iec/4472
Acceso en línea:https://patua.iec.gov.br/handle/iec/4472
Access Level:acceso abierto
Palabra clave:Hepatite C / virologia
Hepacivirus / patogenicidade
Polimorfismo Genético
Fator de Necrose Tumoral alfa
Interferon gama
Interleucina-10
Citocinas
Descripción
Sumario:Background: Genetic changes may induce dysregulated cytokine production and afect the progression of the chronic disease caused by the hepacivirus C (HCV) because the balance of pro- and anti-infammatory cytokines determines the outcome of infection. This study evaluated the TNFA -308G>A and IL10 -1082A>G polymorphisms in the susceptibility and progress of chronic hepatitis C. Method: The study included 101 samples from patients with chronic hepatitis C and 300 samples from healthy donors. Polymorphisms were typed by real-time PCR and were analyzed for associations with histopathological parameters (according to METAVIR classifcation) and HCV viral load. Results: The polymorphic genotype for the TNFA -308G>A variant was not present in the group of patients with chronic hepatitis C and its absence could be associated with protection against HCV infection (p=0.0477). Patients with the polymorphic genotype of the IL10 -1082A>G polymorphism had higher HCV viral load than wild-type patients (p=0.0428). Neither polymorphism was associated with diferent levels of necroinfammatory activity or fbrosis scores. Conclusion: Our results suggest the polymorphic genotype at TNFA -308G>A as protective against chronic HCV infection, and the polymorphic genotype at the IL10 -1082A>G variant associated with higher HCV viral load. Further studies must be performed in order to confrm these associations.