Intracytoplasmic filamentous inclusions and IGHV rearrangements in a patient with chronic lymphocytic leukemia

Chronic lymphocytic leukemia (CLL) represents the most common leukemia in the Western world, accounting for 30-40% of all adult leukemias. The disease is characterized by a highly variable clinical course, ranging from indolent cases to patients with aggressive and rapidly progressing disease. Altho...

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Detalles Bibliográficos
Autores: Rodríguez, Cecilia María, Stanganelli, Carmen Graciela, Bussi, Claudio, Arroyo, Daniela Soledad, Sastre, Darío, Heller, Viviana, Iribarren, Pablo, Slavutsky, Irma Rosa
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2018
País:Argentina
Institución:Consejo Nacional de Investigaciones Científicas y Técnicas
Repositorio:CONICET Digital (CONICET)
Idioma:inglés
OAI Identifier:oai:ri.conicet.gov.ar:11336/63317
Acceso en línea:http://hdl.handle.net/11336/63317
Access Level:acceso abierto
Palabra clave:Cll
Autophagy
Ighv
Leukemia
https://purl.org/becyt/ford/3.2
https://purl.org/becyt/ford/3
Descripción
Sumario:Chronic lymphocytic leukemia (CLL) represents the most common leukemia in the Western world, accounting for 30-40% of all adult leukemias. The disease is characterized by a highly variable clinical course, ranging from indolent cases to patients with aggressive and rapidly progressing disease. Although staging systems are reliable predictors of outcome, they do not fully explain the heterogeneity in treatment response and survival. In the last decades, several prognostic biomarkers have been identified, allowing the subdivision of this heterogeneous disease into clinical relevant subgroups. Among them, genomic alterations and the IGHV (immunoglobulin heavy chain variable region) mutational status are of significance. Particularly, recurrent cytogenetic abnormalities namely deletions of chromosomes 11q, 13q and 17p and, trisomy 12, define subgroups of patients with different clinical behavior and response to treatment while IGHV mutational status permits to distinguish two major CLL subtypes, mutated (M) associated with a good prognosis, and unmutated (UM), characterized by a poor clinical evolution.