Cita APA

Ehmke, N., Hecht, J., & Kornak, U. (2017). De novo mutations in SLC25A24 cause a craniosynostosis syndrome with hypertrichosis, progeroid appearance, and mitochondrial dysfunction.

Citación estilo Chicago

Ehmke, Nadja, Jochen Hecht, y Uwe Kornak. De Novo Mutations in SLC25A24 Cause a Craniosynostosis Syndrome With Hypertrichosis, Progeroid Appearance, and Mitochondrial Dysfunction. 2017.

Cita MLA

Ehmke, Nadja, Jochen Hecht, y Uwe Kornak. De Novo Mutations in SLC25A24 Cause a Craniosynostosis Syndrome With Hypertrichosis, Progeroid Appearance, and Mitochondrial Dysfunction. 2017.

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